Tag Archives: ADHD

How Sugar Affects Your Health – 146 Ways

 

 

(Source: glutenfabulous.org)
(Source: glutenfabulous.org)

 

This list of 146 way sugar affects our health – all detrimental – was compiled by Nancy Appleton, PhD from medical journals and other scientific publications. Dr Appleton is a clinical nutritionist and researcher. She is the author of several books, including Lick The Sugar Habit, Stopping Inflammation: Relieving the Cause of Degenerative Diseases, and Suicide by Sugar: A Startling Look at Our #1 National Addiction. Her website is www.nancyappleton.com

 

1. Sugar can suppress the immune system.

2. Sugar upsets the mineral relationships in the body.

3. Sugar can cause hyperactivity, anxiety, difficulty concentrating, and crankiness in children.

4. Sugar can produce a significant rise in triglycerides.

5. Sugar contributes to the reduction in defense against bacterial infection (infectious diseases).

6. Sugar causes a loss of tissue elasticity and function, the more sugar you eat the more elasticity and function you loose.

7. Sugar reduces high density lipoproteins.

8. Sugar leads to chromium deficiency.

9 Sugar leads to cancer of the ovaries.

10. Sugar can increase fasting levels of glucose.

11. Sugar causes copper deficiency.

12. Sugar interferes with absorption of calcium and magnesium.

13. Sugar can weaken eyesight.

14. Sugar raises the level of a neurotransmitters: dopamine, serotonin, and norepinephrine.

15. Sugar can cause hypoglycemia.

16. Sugar can produce an acidic digestive tract.

17. Sugar can cause a rapid rise of adrenaline levels in children.

18. Sugar malabsorption is frequent in patients with functional bowel disease.

19. Sugar can cause premature aging.

20. Sugar can lead to alcoholism.

21. Sugar can cause tooth decay.

22. Sugar contributes to obesity

23. High intake of sugar increases the risk of Crohn’s disease and ulcerative colitis.

24. Sugar can cause changes frequently found in person with gastric or duodenal ulcers.

25. Sugar can cause arthritis.

26. Sugar can cause asthma.

27. Sugar greatly assists the uncontrolled growth of Candida Albicans (yeast infections).

28. Sugar can cause gallstones.

29. Sugar can cause heart disease.

30. Sugar can cause appendicitis.

31. Sugar can cause multiple sclerosis.

32. Sugar can cause hemorrhoids.

33. Sugar can cause varicose veins.

34. Sugar can elevate glucose and insulin responses in oral contraceptive users.

35. Sugar can lead to periodontal disease.

36. Sugar can contribute to osteoporosis.

37. Sugar contributes to saliva acidity.

38. Sugar can cause a decrease in insulin sensitivity.

39. Sugar can lower the amount of Vitamin E (alpha-Tocopherol in the blood.

40. Sugar can decrease growth hormone.

41. Sugar can increase cholesterol.

42. Sugar can increase the systolic blood pressure.

43. Sugar can cause drowsiness and decreased activity in children.

44. High sugar intake increases advanced glycation end products (AGEs)(Sugar bound non-enzymatically to protein)

45. Sugar can interfere with the absorption of protein.

46. Sugar causes food allergies.

47. Sugar can contribute to diabetes.

48. Sugar can cause toxemia during pregnancy.

49. Sugar can contribute to eczema in children.

50. Sugar can cause cardiovascular disease.

51. Sugar can impair the structure of DNA

52. Sugar can change the structure of protein.

53. Sugar can make our skin age by changing the structure of collagen.

54. Sugar can cause cataracts.

55. Sugar can cause emphysema.

56. Sugar can cause atherosclerosis.

57. Sugar can promote an elevation of low density lipoproteins (LDL).

58. High sugar intake can impair the physiological homeostasis of many systems in the body.

59. Sugar lowers the enzymes ability to function.

60. Sugar intake is higher in people with Parkinson’s disease.

61. Sugar can cause a permanent altering the way the proteins act in the body.

62. Sugar can increase the size of the liver by making the liver cells divide.

63. Sugar can increase the amount of liver fat.

64. Sugar can increase kidney size and produce pathological changes in the kidney.

65. Sugar can damage the pancreas.

66. Sugar can increase the body’s fluid retention.

67. Sugar is enemy #1 of the bowel movement.

68. Sugar can cause myopia (nearsightedness).

69. Sugar can compromise the lining of the capillaries.

70. Sugar can make the tendons more brittle.

71. Sugar can cause headaches, including migraine.

72. Sugar plays a role in pancreatic cancer in women.

73. Sugar can adversely affect school children’s grades and cause learning disorders..

74. Sugar can cause an increase in delta, alpha, and theta brain waves.

75. Sugar can cause depression.

76. Sugar increases the risk of gastric cancer.

77. Sugar and cause dyspepsia (indigestion).

78. Sugar can increase your risk of getting gout.

79. Sugar can increase the levels of glucose in an oral glucose tolerance test over the ingestion of complex carbohydrates.

80. Sugar can increase the insulin responses in humans consuming high-sugar diets compared to low sugar diets.

81 High refined sugar diet reduces learning capacity.

82. Sugar can cause less effective functioning of two blood proteins, albumin, and lipoproteins, which may reduce the body’s ability to handle fat and cholesterol.

83. Sugar can contribute to Alzheimer’s disease.

84. Sugar can cause platelet adhesiveness.

85. Sugar can cause hormonal imbalance; some hormones become underactive and others become overactive.

86. Sugar can lead to the formation of kidney stones.

87. Sugar can lead to the hypothalamus to become highly sensitive to a large variety of stimuli.

88. Sugar can lead to dizziness.

89. Diets high in sugar can cause free radicals and oxidative stress.

90. High sucrose diets of subjects with peripheral vascular disease significantly increases platelet adhesion.

91. High sugar diet can lead to biliary tract cancer.

92. Sugar feeds cancer.

93. High sugar consumption of pregnant adolescents is associated with a twofold increased risk for delivering a small-for-gestational-age (SGA) infant.

94. High sugar consumption can lead to substantial decrease in gestation duration among adolescents.

95. Sugar slows food’s travel time through the gastrointestinal tract.

96. Sugar increases the concentration of bile acids in stools and bacterial enzymes in the colon. This can modify bile to produce cancer-causing compounds and colon cancer.

97. Sugar increases estradiol (the most potent form of naturally occurring estrogen) in men.

98. Sugar combines and destroys phosphatase, an enzyme, which makes the process of digestion more difficult.

99. Sugar can be a risk factor of gallbladder cancer.

100. Sugar is an addictive substance.

101. Sugar can be intoxicating, similar to alcohol.

102. Sugar can exacerbate PMS.

103. Sugar given to premature babies can affect the amount of carbon dioxide they produce.

104. Decrease in sugar intake can increase emotional stability.

105. The body changes sugar into 2 to 5 times more fat in the bloodstream than it does starch.

106. The rapid absorption of sugar promotes excessive food intake in obese subjects.

107. Sugar can worsen the symptoms of children with attention deficit hyperactivity disorder (ADHD).

108. Sugar adversely affects urinary electrolyte composition.

109. Sugar can slow down the ability of the adrenal glands to function.

110. Sugar has the potential of inducing abnormal metabolic processes in a normal healthy individual and to promote chronic degenerative diseases.

111.. IVs (intravenous feedings) of sugar water can cut off oxygen to the brain.

112. High sucrose intake could be an important risk factor in lung cancer.

113. Sugar increases the risk of polio.

114. High sugar intake can cause epileptic seizures.

115. Sugar causes high blood pressure in obese people.

116. In Intensive Care Units, limiting sugar saves lives.

117. Sugar may induce cell death.

118. Sugar can increase the amount of food that you eat.

119. In juvenile rehabilitation camps, when children were put on a low sugar diet, there was a 44% drop in antisocial behavior.

120. Sugar can lead to prostate cancer.

121. Sugar dehydrates newborns.

122. Sugar increases the estradiol in young men.

123. Sugar can cause low birth weight babies.

124. Greater consumption of refined sugar is associated with a worse outcome of schizophrenia

125. Sugar can raise homocysteine levels in the blood stream.

126. Sweet food items increase the risk of breast cancer.

127. Sugar is a risk factor in cancer of the small intestine.

128. Sugar may cause laryngeal cancer.

129. Sugar induces salt and water retention.

130. Sugar may contribute to mild memory loss.

131. As sugar increases in the diet of 10 years olds, there is a linear decrease in the intake of many essential nutrients.

132. Sugar can increase the total amount of food consumed.

133. Exposing a newborn to sugar results in a heightened preference for sucrose relative to water at 6 months and 2 years of age.

134. Sugar causes constipation.

135. Sugar causes varicose veins.

136. Sugar can cause brain decay in prediabetic and diabetic women.

137. Sugar can increase the risk of stomach cancer.

138. Sugar can cause metabolic syndrome.

139. Sugar ingestion by pregnant women increases neural tube defects in embryos.

140. Sugar can be a factor in asthma.

141. The higher the sugar consumption the more chances of getting irritable bowel syndrome.

142. Sugar could affect central reward systems.

143. Sugar can cause cancer of the rectum.

144. Sugar can cause endometrial cancer.

145. Sugar can cause renal (kidney) cell carcinoma.

146. Sugar can cause liver tumors.

 

 

inflammationlr1

 

 

 

imgres

 

Many thanks to Dr Beth Forgosh, of Discover Chiropractic of Soho, for bringing Dr Appleton’s list to my attention.

 

 

Note added to this post on 12/29/2014:

 

fruit-vs-dessert

 

Suzette Lawrence, MSN, commented that Dr Appleton’s list, above, describes the effects of REFINED sugars:

“This is not the case for natural fruits sugars that are attached to the fiber in the fruit, known as levulose … if absorbed it occurs low in the intestines and is converted to glycogen in the liver and stored there as an emergency energy source.  I agree that the SAD (Standard American Diet) beginning in infancy sets the stage for every disease, and some new ones. Think, GMO beet sugar … ”

From a 2014 article by the Cancer Treatment Centers of America entitled Natural vs. refined sugars – What’s the difference?:

Sugar, in all forms, is a simple carbohydrate that the body converts into glucose and uses for energy. But the effect on the body and your overall health depends on the type of sugar you’re eating, either natural or refined.

We wanted to explore the difference between these sugar types as a follow-up to our post about whether sugar drives the growth of cancer, which has received several comments. We again turned to Julie Baker, Clinical Oncology Dietitian at our hospital outside Atlanta, for her expertise on the issue.

Understanding sugars

Natural sugars are found in fruit as fructose and in dairy products, such as milk and cheese, as lactose. Foods with natural sugar have an important role in the diet of cancer patients and anyone trying to prevent cancer because they provide essential nutrients that keep the body healthy and help prevent disease.

Refined sugar comes from sugar cane or sugar beets, which are processed to extract the sugar. It is typically found as sucrose, which is the combination of glucose and fructose. We use white and brown sugars to sweeten cakes and cookies, coffee, cereal and even fruit. Food manufacturers add chemically produced sugar, typically high-fructose corn syrup, to foods and beverages, including crackers, flavored yogurt, tomato sauce and salad dressing. Low-fat foods are the worst offenders, as manufacturers use sugar to add flavor.

Most of the processed foods we eat add calories and sugar with little nutritional value. In contrast, fruit and unsweetened milk have vitamins and minerals. Milk also has protein and fruit has fiber, both of which keep you feeling full longer.

DR APPLETON’S REFERENCES

1. Sanchez, A., et al. Role of Sugars in Human Neutrophilic Phagocytosis, American Journal of Clinical Nutrition. Nov 1973;261:1180-1184.
Bernstein, J., et al. Depression of Lymphocyte Transformation Following Oral Glucose Ingestion. American Journal of Clinical Nutrition.1997;30:613.
2. Couzy, F., et al. Nutritional Implications of the Interaction Minerals, Progressive Food and Nutrition Science 17;1933:65-87.
3. Goldman, J., et al. Behavioral Effects of Sucrose on Preschool Children. Journal of Abnormal Child Psychology. 1986;14(4):565-577.
4. Scanto, S. and Yudkin, J. The Effect of Dietary Sucrose on Blood Lipids, Serum Insulin, Platelet Adhesiveness and Body Weight in Human Volunteers, Postgraduate Medicine Journal. 1969;45:602-607.
5. Ringsdorf, W., Cheraskin, E. & Ramsay R. Sucrose,Neutrophilic Phagocytosis and Resistance to Disease, Dental Survey. 1976;52(12):46-48.
6. Cerami, A., Vlassara, H., & Brownlee, M. Glucose and Aging. Scientific American. May 1987:90.
Lee, A. T. & Cerami, A. The Role of Glycation in Aging. Annals of the New York Academy of Science. 663:63-67.
7. Albrink, M. & Ullrich I. H. Interaction of Dietary Sucrose and Fiber on Serum Lipids in Healthy Young Men Fed High Carbohydrate Diets. American Journal of Clinical Nutrition. 1986;43:419-428.
Pamplona, R., et al. Mechanisms of Glycation in Atherogenesis. Medical Hypotheses. Mar 1993;40(3):174-81.
8. Kozlovsky, A., et al. Effects of Diets High in Simple Sugars on Urinary Chromium Losses. Metabolism. June 1986;35:515-518.
9. Takahashi, E., Tohoku University School of Medicine, Holistic Health Digest. October 1982:41.
10. Kelsay, J., et al. Diets High in Glucose or Sucrose and Young Women. American Journal of Clinical Nutrition. 1974;27:926-936.
Thomas, B. J., et al. Relation of Habitual Diet to Fasting Plasma Insulin Concentration and the Insulin Response to Oral Glucose. Human Nutrition Clinical Nutrition. 1983; 36C(1):49_51.
11. Fields, M., et al. Effect of Copper Deficiency on Metabolism and Mortality in Rats Fed Sucrose or Starch Diets, Journal of Clinical Nutrition. 1983;113:1335-1345.
12. Lemann, J. Evidence that Glucose Ingestion Inhibits Net Renal Tubular Reabsorption of Calcium and Magnesium. Journal Of Clinical Nutrition. 1976 ;70:236-245.
13. Acta Ophthalmologica Scandinavica. Mar 2002;48;25.
Taub, H. Ed. Sugar Weakens Eyesight, VM NEWSLETTER; May 1986:6
14. Sugar, White Flour Withdrawal Produces Chemical Response. The Addiction Letter. Jul 1992:4.
15. Dufty, William. Sugar Blues. (New York:Warner Books, 1975).
16. Ibid.
17. Jones, T. W., et al. Enhanced Adrenomedullary Response and Increased Susceptibility to Neuroglygopenia: Mechanisms Underlying the Adverse Effect of Sugar Ingestion in Children. Journal of Pediatrics. Feb 1995;126:171-7.
18. Ibid.
19. Lee, A. T. & Cerami A. The Role of Glycation in Aging.” Annals of the New York Academy of Science.1992;663:63-70.
20. Abrahamson, E. & Peget, A. Body, Mind and Sugar. (New York:Avon,1977.}
21. Glinsmann, W., Irausquin, H., & Youngmee, K. Evaluation of Health Aspects of Sugar Contained in Carbohydrate Sweeteners. F. D. A. Report of Sugars Task Force. 1986:39.
Makinen K.K.,et al. A Descriptive Report of the Effects of a 16-month Xylitol Chewing-Gum Programme Subsequent to a 40-Month Sucrose Gum Programme. Caries Research. 1998; 32(2)107-12.
Riva Touger-Decker & Cor van Loveren, Sugars and Dental Caries.
American Journal of Clinical Nutrition. Oct 2003; 78:881-892.
22. Keen, H., et al. Nutrient Intake, Adiposity, and Diabetes. British Medical Journal. 1989; 1: 655-658.
23. Tragnone, A. et al. Dietary Habits as Risk Factors for Inflammatory Bowel Disease. European Journal of Gastroenterological Hepatology. Jan 1995;7(1):47-51.
24. Yudkin, J. Sweet and Dangerous. (New York;Bantam Books:1974), 129.
25. Darlington, L., Ramsey, N. W. & Mansfield, J. R. Placebo_Controlled, Blind Study of Dietary Manipulation Therapy in Rheumatoid Arthritis, Lancet. Feb 1986;8475(1):236-238.
26. Powers, L. Sensitivity: You React to What You Eat. Los Angeles Times. Feb. 12, 1985.
Cheng, J., et al. Preliminary Clinical Study on the Correlation Between Allergic Rhinitis and Food Factors. Lin Chuang Er Bi Yan Hou Ke Za Zhi Aug 2002;16(8):393-396.
27. Crook, W. J. The Yeast Connection. (TN:Professional Books, 1984)..
28. Heaton, K. The Sweet Road to Gallstones. British Medical Journal. Apr 14, 1984; 288:1103-1104.
Misciagna, G., et al. American Journal of Clinical Nutrition. 1999;69:120-126.
29. Yudkin, J. Sugar Consumption and Myocardial Infarction. Lancet.Feb 6, 1971;1(7693):296-297.
Reiser, S. Effects of Dietary Sugars on Metabolic Risk Factors Associated with Heart Disease. Nutritional Health. 1985;203-216.
30. Cleave, T. The Saccharine Disease. (New Canaan, CT: Keats Publishing, 1974).
31. Erlander, S. The Cause and Cure of Multiple Sclerosis, The Disease to End Disease. Mar 3, 1979;1(3):59-63.
32. Cleave, T. The Saccharine Disease. (New Canaan, CT: Keats Publishing, 1974.)
33. Cleave, T. & Campbell, G. Diabetes, Coronary Thrombosis and the Saccharine Disease. (Bristol, England, John Wrightand Sons, 1960).
34. Behall, K. Influence of Estrogen Content of Oral Contraceptives and Consumption of Sucrose on Blood Parameters. Disease Abstracts International. 1982;431-437.
35. Glinsmann, W., Irausquin, H., & K. Youngmee. Evaluation of Health Aspects of Sugar Contained in Carbohydrate Sweeteners. F. D. A. Report of Sugars Task Force. 1986;39:36_38.
36. Tjderhane, L. & Larmas, M. A High Sucrose Diet Decreases the Mechanical Strength of Bones in Growing Rats. Journal of Nutrition. 1998:128:1807-1810.
37. Appleton, N. Healthy Bones. New York: Avery Penguin Putnam,1989.
38. Beck_Nielsen H., Pedersen O., & Schwartz S. Effects of Diet on the Cellular Insulin Binding and the Insulin Sensitivity in Young Healthy Subjects. Diabetes. 1978;15:289-296 .
39. Mohanty P. et al. Glucose Challenge Stimulates Reactive Oxygen Species (ROS) Generation by Leucocytes. Journal of Clinical Endocrinology and Metabolism. Aug 2000; 85(8):2970-2973.
40. Gardner, L. & Reiser, S. Effects of Dietary Carbohydrate on Fasting Levels of Human Growth Hormone and Cortisol. Proceedings of the Society for Experimental Biology and Medicine. 1982;169:36-40.
41. Reiser, S. Effects of Dietary Sugars on Metabolic Risk Factors Associated with Heart Disease. Nutritional Health. 1985;203:216.
42. Preuss, H. G. Sugar-Induced Blood Pressure Elevations Over the Lifespan of Three Substrains of Wistar Rats. Journal of the American College of Nutrition, 1998;17(1) 36-37.
43. Behar, D., et al. Sugar Challenge Testing with Children Considered Behaviorally Sugar Reactive. Nutritional Behavior. 1984;1:277-288.
44. Furth, A. & Harding, J. Why Sugar Is Bad For You. New Scientist. Sep 23, 1989;44.
45. Lee AT, & Cerami A. Role of Glycation in Aging. Annals of the New York Academy of Science. Nov 21,1992 ;663:63-70.
46. Appleton, N. Lick the Sugar Habit. (New York:Avery Penguin Putnam:1988).
47. Sucrose Induces Diabetes in Cats. Federal Protocol. 1974;6(97).
48. Cleave, T. The Saccharine Disease (New Canaan Ct: Keats Publishing, Inc., 1974).131.
49. Ibid. 132.
50. Vaccaro O., Ruth, K. J. & Stamler J. Relationship of Postload Plasma Glucose to Mortality with 19 Year Follow-up. Diabetes Care. Oct 15,1992;10:328-334.
Tominaga, M., et al, Impaired Glucose Tolerance Is a Risk Factor for Cardiovascular Disease, but Not Fasting Glucose. Diabetes Care. 1999:2(6):920-924.
51. Lee, A. T. & Cerami, A. Modifications of Proteins and Nucleic Acids by Reducing Sugars: Possible Role in Aging. Handbook of the Biology of Aging. (New York: Academic Press, 1990.).
52. Monnier, V. M. Nonenzymatic Glycosylation, the Maillard Reaction and the Aging Process. Journal of Gerontology. 1990:45(4 ):105-110.
53. Dyer, D. G., et al. “=Accumulation of Maillard Reaction Products in Skin Collagen in Diabetes and Aging. Journal of Clinical Investigation. 1993:93(6):421-422.
54. Veromann, S.et al. Dietary Sugar and Salt Represent Real Risk Factors for Cataract Development. Ophthalmologica. Jul-Aug 2003 ;217(4):302-307.
55. Monnier, V. M. Nonenzymatic Glycosylation, the Maillard Reaction and the Aging Process. Journal of Gerontology. 1990:45(4):105-110.
56. Schmidt A.M. et al. Activation of receptor for advanced glycation end products: a mechanism for chronic vascular dysfunction in diabetic vasculopathy and atherosclerosis. Circular Research Archives. 1999 Mar 19;84(5):489-97.
57. Lewis, G. F. and Steiner, G. Acute Effects of Insulin in the Control of VLDL Production in Humans. Implications for Theinsulin-resistant State. Diabetes Care. 1996 Apr;19(4):390-3
R. Pamplona, M. .J., et al. Mechanisms of Glycation in Atherogenesis. Medical Hypotheses. 1990;40:174-181.
58. Ceriello, A. Oxidative Stress and Glycemic Regulation. Metabolism. Feb 2000;49(2 Suppl 1):27-29.
59. Appleton, Nancy. Lick the Sugar Habit. (New York:Avery Penguin Putnam, 1988).
60. Hellenbrand, W. Diet and Parkinson’s Disease. A Possible Role for the Past Intake of Specific Nutrients. Results from a Self-administered Food-frequency Questionnaire in a Case-control Study. Neurology. Sep 1996;47(3):644-650 Cerami, A., Vlassara, H., & Brownlee, M. Glucose and Aging. Scientific American. May 1987: 90.
62. Goulart, F. S. Are You Sugar Smart? American Fitness. Mar-Apr 1991: 34-38.
63. Ibid.
64. Yudkin, J., Kang, S. & Bruckdorfer, K. Effects of High Dietary Sugar. British Journal of Medicine. Nov 22, 1980;1396.
65. Goulart, F. S. Are You Sugar Smart? American Fitness. March_April 1991: 34-38
66. Ibid.
67. Ibid.
68. Ibid.
69. Ibid.
70. Nash, J. Health Contenders. Essence. Jan 1992-23: 79_81.
71. Grand, E. Food Allergies and Migraine. Lancet. 1979:1:955_959.
72. Michaud, D. Dietary Sugar, Glycemic Load, and Pancreatic Cancer Risk in a Prospective Study. Journal of the National Cancer Institute. Sep 4, 2002 ;94(17):1293-300.
73. Schauss, A. Diet, Crime and Delinquency. (Berkley Ca; Parker House, 1981).
74. Christensen, L. The Role of Caffeine and Sugar in Depression. Nutrition Report. Mar 1991;9(3):17-24.
75. Ibid.
76. Cornee, J., et al. A Case-control Study of Gastric Cancer and Nutritional Factors in Marseille, France, European Journal of Epidemiology. 1995;11:55-65.
77. Yudkin, J. Sweet and Dangerous.(New York:Bantam Books,1974) 129.
78. Ibid, 44
79. Reiser, S., et al. Effects of Sugars on Indices on Glucose Tolerance in Humans. American Journal of Clinical Nutrition. 1986:43;151-159.
80. Reiser,S., et al. Effects of Sugars on Indices on Glucose Tolerance in Humans.  American Journal of Clinical Nutrition. 1986;43:151-159.
81. Molteni, R, et al. A High-fat, Refined Sugar Diet Reduces Hippocampal Brain-derived Neurotrophic Factor, Neuronal Plasticity, and Learning. NeuroScience. 2002;112(4):803-814.
82. Monnier, V., Nonenzymatic Glycosylation, the Maillard Reaction and the Aging Process. Journal of Gerontology. 1990;45:105-111.
83. Frey, J. Is There Sugar in the Alzheimers Disease? Annales De Biologie Clinique. 2001; 59 (3):253-257.
84. Yudkin, J. Metabolic Changes Induced by Sugar in Relation to Coronary Heart Disease and Diabetes. Nutrition and Health. 1987;5(1-2):5-8.
85. Ibid.
86. Blacklock, N. J., Sucrose and Idiopathic Renal Stone. Nutrition and Health. 1987;5(1-2):9-12.
Curhan, G., et al. Beverage Use and Risk for Kidney Stones in Women. Annals of Internal Medicine. 1998:28:534-340.
87. Journal of Advanced Medicine. 1994;7(1):51-58.
88. Ibid.
89. Ceriello, A. Oxidative Stress and Glycemic Regulation. Metabolism. Feb 2000;49(2 Suppl 1):27-29.
90. Postgraduate Medicine. Sept 1969:45:602-07.
91. Moerman, C. J., et al. Dietary Sugar Intake in the Etiology of Biliary Tract Cancer. International Journal of Epidemiology. Ap 1993;2(2):207-214.
92. Quillin, Patrick, Cancer’s Sweet Tooth. Nutrition Science News. Apr 2000.
Rothkopf, M.. Nutrition. July/Aug 1990;6(4).
93. Lenders, C. M. Gestational Age and Infant Size at Birth Are Associated with Dietary Intake among Pregnant Adolescents. Journal of Nutrition. Jun 1997;1113-1117.
94. Ibid.
95. Bostick, R. M., et al. Sugar, Meat and Fat Intake and Non-dietary Risk Factors for Colon Cancer Incidence in Iowa Women. Cancer Causes & Control. 1994:5:38-53.
96. Ibid.
Kruis, W., et al. Effects of Diets Low and High in Refined Sugars on Gut Transit, Bile Acid Metabolism and Bacterial Fermentation. Gut. 1991;32:367-370.
Ludwig, D. S., et al. High Glycemic Index Foods, Overeating, And Obesity. Pediatrics. Mar 1999;103(3):26-32.
97. Yudkin, J. & Eisa, O. Dietary Sucrose and Oestradiol Concentration in Young Men. Annals of Nutrition and Metabolism. 1988:32(2):53-55.
98. Lee, A. T. & Cerami A. The Role of Glycation in Aging. Annals of the New York Academy of Science. 1992; 663:63-70.
99. Moerman, C. et al. Dietary Sugar Intake in the Etiology of Gallbladder Tract Cancer. International Journal of Epidemiology. Apr 1993; 22(2):207-214.
100. Sugar, White Flour Withdrawal Produces Chemical Response. The Addiction Letter. Jul 1992:4.
Colantuoni, C., et al. Evidence That Intermittent, Excessive Sugar Intake Causes Endogenous Opioid Dependence. Obesity Research. Jun 2002 ;10(6):478-488.
101. Ibid.
102. The Edell Health Letter. Sept 1991;7:1.
103. Sunehag, A. L., et al. Gluconeogenesis in Very Low Birth Weight Infants Receiving Total Parenteral Nutrition. Diabetes. 1999 ;48 7991-8000).
104. Christensen L. et al. Impact of A Dietary Change on Emotional Distress. Journal of Abnormal Psychology. 1985;94(4):565-79.
105. Nutrition Health Review. Fall 85. Sugar Changes into Fat Faster than Fat.
106. Ludwig, D. S., et al. High Glycemic Index Foods, Overeating and Obesity. Pediatrics. Mar 1999;103(3):26-32.
107. Girardi, N.L. Blunted Catecholamine Responses after Glucose Ingestion in Children with Attention Deficit Disorder. Pediatrics Research. 1995;38:539-542.
Berdonces, J. L. Attention Deficit and Infantile Hyperactivity. Rev Enferm. Jan 2001;4(1)11-4
108. Blacklock, N. J. Sucrose and Idiopathic Renal Stone. Nutrition Health. 1987;5(1 & 2):9-17.
109. Lechin, F., et al. Effects of an Oral Glucose Load on Plasma Neurotransmitters in Humans. Neurophychobiology. 1992;26(1-2):4-11.
110. Fields, M. Journal of the American College of Nutrition. Aug 1998;17(4):317-321.
111. Arieff, A. I. Veterans Administration Medical Center in San Francisco. San Jose Mercury. June 12/86. IVs of Sugar Water Can Cut Off Oxygen to the Brain.
112. De Stefani, E.Dietary Sugar and Lung Cancer: a Case Control Study in Uruguay. Nutrition and Cancer. 1998;31(2):132_7.
113. Sandler, Benjamin P. Diet Prevents Polio. Milwakuee, WI,:The Lee Foundation for for Nutritional Research, 1951.
114. Murphy, Patricia. The Role of Sugar in Epileptic Seizures. Townsend Letter for Doctors and Patients. May, 2001.
115. Stern, N. & Tuck, M. Pathogenesis of Hypertension in Diabetes Mellitus. Diabetes Mellitus, a Fundamental and Clinical Test. 2nd Edition, (Phil. A: Lippincott Williams & Wilkins, 2000)943-957.
116. Christansen, D. Critical Care: Sugar Limit Saves Lives. Science News. June 30, 2001;159:404.
117. Donnini, D. et al. Glucose May Induce Cell Death through a Free Radical-mediated Mechanism.Biochem Biohhys Res Commun. Feb 15, 1996:219(2):412-417.
118. Allen S. Levine, Catherine M. Kotz, & Blake A. Gosnell . Sugars and Fats: The Neurobiology of Preference. Journal of Nutrition. 2003 133:831S-834S.
119. Schoenthaler, S. The Los Angeles Probation Department Diet-Behavior Program: An Empirical Analysis of Six Institutional Settings. International Journal of Biosocial Research. 5(2):88-89.
120. Deneo-Pellegrini H,. et al. Foods, Nutrients and Prostate cancer: a Case-control study in Uruguay. Br J Cancer. 1999 May;80(3-4):591-7.
121. Gluconeogenesis in Very Low Birth Weight Infants Receiving Total Parenteral Nutrition. Diabetes. 1999 Apr;48(4):791-800.
122. Yudkin, J. and Eisa, O. Dietary Sucrose and Oestradiol Concentration in Young Men. Annals of Nutrition and Metabolism. 1988;32(2):53-5.
123. Lenders, C. M. Gestational Age and Infant Size at Birth Are Associated with Dietary Intake Among Pregnant Adolescents. Journal of Nutrition.128; 1998::807-1810.
124. Peet, M. International Variations in the Outcome of Schizophrenia and the Prevalence of Depression in Relation to National Dietary Practices: An Ecological
Analysis. British Journal of Psychiatry. 2004;184:404-408.
125. Fonseca, V. et al. Effects of a High-fat-sucrose Diet on Enzymes in Homosysteine Metabolism in the Rat. Metabolism. 200; 49:736-41.
126. Potischman, N, et.al. Increased Risk of Early-stage Breast Cancer Related to Consumption of Sweet Foods among Women Less than Age 45 in the United States. Cancer Causes Control. 2002 Dec;13(10):937-46.
127.Negri. E. et al. Risk Factors for Adenocarcinoma of the Small Intestine.
International Journal of Cancer. 1999:82:I2:171-174.
128.Bosetti, C. et al. Food Groups and Laryngeal Cancer Risk: A Case-control Study from Italy and Switzerland. International Journal of Cancer, 2002:100(3): 355-358.
129. Shannon, M. An Empathetic Look at Overweight.CCL Family Foundation. Nov-Dec.1993. 20(3):3-5.
130. Harry G. Preuss, MD, of Georgetown University Medical School.
131. Health After 50. Johns Hopkins Medical Letter. May, 1994.
132. Allen, S. Sugars and Fats: The Neurobiology of Preference. Journal of Nutrition. 2003;133:831S-834S.
133. Booth, D.A.M. et al. Sweetness and Food Selection: Measurement of Sweeteners Effects on Acceptance. Sweetness. Dobbing, J., Ed., (London:Springer-Verlag, 1987).
134. Cleve, T.L On the Causation of Varicose Veins. Bristol, England, John Wright, 1960.
135. Cleve, T.L On the Causation of Varicose Veins. Bristol, England, John Wright, 1960.
136. Ket, Yaffe et al. Diabetes, Impaired Fasting Glucose and Development of Cognitive Impairment in Older Women. Neurology. 2004;63:658�663.
137. Chatenoud, Liliane et al. Refined-cereal Intake and Risk of Selected Cancers in Italy. American Journal of Clinical Nutrition, Dec 1999;70:1107-1110.
138. Yoo, Sunmi et al. Comparison of Dietary Intakes Associated with Metabolic Syndrome Risk Factors in Young Adults: the Bogalusa Heart Study. American Journal of Clinical Nutrition.  2004 Oct;80(4):841-848.
139. Shaw, Gary M. et al. Neural Tube Defects Associated with Maternal Periconceptional Dietary Intake of Simple Sugars and Glycemic Index.
American Journal of Clinical Nutrition, Nov 2003;78:972-978.
140. Krilanovich, Nicholas J. Fructose Misuse, the Obesity Epidemic, the Special Problems of the Child, and a Call to Action  American Journal of Clinical Nutrition, Nov 2004;80:1446-1447.
141.Jarnerot, G., Consumption of Refined Sugar by Patients with Crohn’s Disease, Ulcerative colitis, or Irritable Bowel Syndrome. Scandinavian Journal of Gastroenterology. 1983 Nov;18(8):999-1002.
142. Allen, S. Sugars and Fats: The Neurobiology of Preference. Journal of Nutrition.
2003;133:831S-834S.
143. De Stefani E, Mendilaharsu M, & Deneo-Pellegrini H. Sucrose as a Risk Factor for Cancer of the Colon and Rectum: a Case-control Study in Uruguay. International Journal of Cancer. 1998 Jan 5;75(1):40-4.
144. Levi F, Franceschi S, Negri E, & La Vecchia C. Dietary Factors and the Risk of Endometrial Cancer. Cancer. 1993 Jun 1;71(11):3575-3581.
145. Mellemgaard A. et al. Dietary Risk Factors for Renal Cell Carcinoma in Denmark. European Journal of Cancer. 1996 Apr;32A(4):673-82.
146. Rogers AE, Nields HM, & Newberne PM. Nutritional and Dietary Influences on Liver Tumorigenesis in Mice and Rats. Arch Toxicol Suppl. 1987;10:231-43. Review.

 

 

© Copyright 2014 Joan Rothchild Hardin. All Rights Reserved.

 

DISCLAIMER:  Nothing on this site or blog is intended to provide medical advice, diagnosis or treatment.

Food Additives and Hyperactivity in Children

 

 

(Source: www.healthy-holistic-living.com)
(Source: www.healthy-holistic-living.com)

 

The US Food and Drug Administration (FDA) has approved OVER 70,000 FOOD ADDITIVES  –  from artificial ingredients to genetically modified ones, and from all natural to GRAS (generally recognized as safe). The situation is even worse with cosmetics and personal care products, with virtually no regulations on chemicals. Our skin absorbs whatever we put on it so those chemicals get into our bodies, where many of them do harm. (HealthyHolisticLiving.com, 2014)
Does this alarm you?

 

 

 

Europe Bans Food Dyes due to ADHD & Cancer Links. (Source: csglobe.com)
Europe Bans Food Dyes due to ADHD & Cancer Links – US FDA Continues to Allow Them. (Source: csglobe.com)

 

 

When British researchers removed food additives from the diets of a group of hyperactive 3-year-olds, the children calmed down. When artificial food colorings and preservatives were introduced back into the children’s diets, their parents reported an increase in hyperactivity. Researchers estimate that if children were to eat additive-free, the number of them thought to have hyperactivity-related behavioral problems could be greatly reduced.  (PureVolume.com, 2014)

 

 

 

 

THE US FDA’S LAX REGULATION OF FOOD ADDITIVES (AllGov.com, 2014) (Thompson, 2013)

 

 

images-4

 

The Natural Resources Defense Council (NDRC) reviewed the food safety protection system managed by the US Food and Drug Administration (FDA) and concluded that Americans are consuming food laced with unsafe chemicals due to our federal government’s ongoing failure to oversee and adequately regulate food producers.

“Rules governing the chemicals that go into a tennis racket are more stringent than (rules for) the chemicals that go into our food. At least when you put a new chemical on the market, you have to notify the EPA (Environmental Protection Agency). But there’s no requirement that you notify the FDA when you make a new food additive.”

– Thomas Nelter, J.D., Director of the Pew Charitable Trust’s Food Additives Project

Mr Nelter is a Chemical Engineer, an Attorney, a Healthy Homes specialist, and a Fellow of the Institute of Hazardous Materials Management.

 

 

Thomas Nelter, Head of the Pew Charitable Trust (Source: iflr.foodlaw.org)
Thomas Nelter, Director of the Pew Charitable Trust’s Food Additives Project.  (Source: iflr.foodlaw.org)
Nelter’s project studied conflict of interest issues in food safety evaluations and found that, between 1997-2012, employees of food additive manufacturers wrote 1 of every 5 safety determinations the industry submitted to the US FDA.
Another 12% of the safety determinations were paid for by a consulting firm selected by the manufacturer. The rest of the safety reviews were conducted by expert panels chosen either by the manufacturer or by a consultant to the manufacturer.
According to an article published in the respected journal JAMA Internal Medicine, another 13% of the determinations were written by someone working for a consulting firm selected by the manufacturer and the remainder of the reviews were conducted by expert panels selected either by the manufacturer or a consultant to the manufacturer.
Hmm, this does not seem like a good way to protect the public
The Pew study employed conflict of interest criteria developed by a committee of the Institute of Medicine to analyze 451generally recognized as safe, or GRAS, determinations that the food industry submitted to the US FDA over a 25 year period.
The Food Additives Amendment of 1958 authorizes MANUFACTURERS to make determinations of an additive’s safety (GRAS determinations) but doesn’t require them to notify the FDA about these determinations.

 

Really?
Really?

 

 

 

 

 

FOOD DYES, ADHD, CANCER AND ALLERGIES (Curran, 2010)

So many vibrantly colored products – I hesitate to call them foods – are available in the processed foods aisles we may not give their colors a second thought – yet we should because the FDA-approved food dyes used to achieve those colors present profound risks to our health. The Center for Science in the Public Interest (CSPI)  published FOOD DYES; A RAINBOW OF RISKS, a comprehensive scientific report detailing the risks from nine dyes widely used in food products. The report was compiled by Molecular Toxicologist Sarah Kobylewski and Michael Jacobson, Executive Director of the Center for Science in the Public Interest.
This report presents considerable evidence for risks of cancer, hyperactivity in children and allergies posed by food dyes.
THE US FOOD INDUSTRY POURS INTO OUR FOOD SUPPLY OVER 15 MILLION POUNDS OF THE NINE STUDIED DYES – PER YEAR. Three of these dyes contain known carcinogens. Four can cause serious allergic reactions. Other studies have found that seven of the nine contributed to cancer development in lab animals, including brain and testicular tumors, colon cancer, and mutations.

 

CSPI’s Executive Director, Michael F. Jacobson, said, “These synthetic chemicals do absolutely nothing to improve the nutritional quality or safety of foods, but trigger behavior problems in children and, possibly, cancer in anybody.”
An Associate at the National Toxicology Program, James Huff,  commented, “Some dyes have caused cancers in animals, contain cancer-causing contaminants, or have been inadequately tested for cancer or other problems. Their continued use presents unnecessary risks to humans, especially young children. It’s disappointing that the [U.S. Food and Drug Administration] has not addressed the toxic threat posed by food dyes.”
A letter CSPI mailed to the FDA detailed reasons for banning food dyes in the US. to protect consumers.  CSPI charged that the FDA fails to enforce the law in the following ways:
  • Red 3 and Citrus Red 2 should be banned under the Delaney amendment, because they caused cancer in rats (some uses were banned in 1990), as should Red 40, Yellow 5, and Yellow 6, which are tainted with cancer-causing contaminants.
  • Evidence suggests, though does not prove, that Blue 1, Blue 2, Green 3, Red 40 and Yellow 6 cause cancer in animals. There is certainly not “convincing evidence” of safety.
  • Dyed foods should be considered adulterated under the law, because the dyes make a food “appear better or of greater value than it is” – typically by masking the absence of fruit, vegetable or other more costly ingredient.”
CSPI charged that the FDA has been aware of  the health risks posed by these dyes but has not acted to protect consumers.
As of 2010, when the CSPI report was issued, over 200,000 pounds of Red 3 was added to  processed food products each year, including ConAgra’s Kid Cuisine frozen meals and Betty Crocker’s Fruit Roll-Ups. (I don’t know the statistics for the years after 2010.)
If food manufacturers wish to attract consumers with food colors, they have choices other than synthetic, petroleum-based dyes: Blueberry juice concentrate, carrot juice, paprika, grape skin extract, beet juice, purple sweet potato, corn, and red cabbage are just a few of the many non-chemical dyes available – and used in other countries.
Fanta Orange Soda manufactured in Britain is colored with pumpkin and carrot extract while the US version is dyed with Red 40 and Yellow 6. Kellogg Strawberry NutriGrain Bars are colored with Red 40, Yellow 6 and Blue 1 in the US but with beetroot, annatto and paprika extract in Britain. McDonald’s Strawberry Sundaes are colored Red dye 40 in America but with strawberries in Britain.
American consumer advocacy groups have called on the FDA to enact similar policies in the this country.
A British law went into effect on July 20th 2010 requiring companies to put this notice on each dyed product sold in Europe:  “May have an adverse effect on activity and attention in children.”

 

 

 

 

IS OUR FDA TRULY PROTECTING US?

 

(Source; www.change.org)
(Source; www.change.org)
The images below show coloring agents used in Kellogg’s Nutri-Grain bars made in the US vs in Britain.  In Britain, lawmakers believe potentially harmful ingredients should be banned from foods until they’re proven safe. In the US, our FDA takes the position that these ingredients can be left in until they have been proven to be harmful. From my research, it seems even when they’ve been scientifically determined to be harmful, the FDA often refuses to ban them. GMO foods are a case in point.
(Source: www.drfranklipman.com)
(Source: www.drfranklipman.com)

 

 

 

 

 

NINE FOOD ADDITIVES ASSOCIATED WITH HYPERACTIVITY

 

Here’s a list of nine food additives that could aggravate attention problems (Gardner, 2014):

 

BLUE NO.1: A food coloring (AKA Brilliant Blue)

Found in:

Frito-Lay Sun Chips French Onion and other Frito-Lay products; some Yoplait products; some JELL-O dessert products; Fruity Cheerios; Trix; Froot-Loops; Apple Jacks; Quaker Cap’N Crunch’s Crunch Berries; some Pop-Tarts products; some Oscar Mayer Lunchables; Duncan Hines Whipped Frosting Chocolate; Edy’s ice cream products; Skittles candies; Jolly Ranchers Screaming Sours Soft & Chew Candy; Eclipse gum; Fanta Grape

Some products containing FDA-approved Blue 1
Some products containing FDA-approved Blue No. 1

 

 

 

Blue No. 2: A food coloring (AKA Indigotine)

Found in:
Froot-Loops; Post Fruity Pebbles; Pop-Tarts products; Duncan Hines Moist Deluxe Strawberry Supreme Premium Cake Mix; Betty Crocker Frosting Rich & Creamy Cherry; M&M’s Milk Chocolate Candies; M&M’s Milk Chocolate Peanut Candies; Wonka Nerds Grape/Strawberry; pet foods

 

Some food products containing FDA-approved Blue No. 2
Some food products containing FDA-approved Blue No. 2

 

 

 

Green No.3: A food coloring, rarely used now

Found in:

Candy, beverages, ice cream, puddings

 

Example of food product containing FDA-approved Green No. 3
Example of food product containing FDA-approved Green No. 3

 

 

 

Orange B: A food coloring that used to added to sausage casings

 

FDA-approved Orange B in sausage casings
FDA-approved Orange B in sausage casings

Red No.3: A food coloring (AKA Carmoisine)

Found in:

Candy, cake icing, chewing gum

 

Examples of food products containing FDA-approved Red No. 3
Examples of food products containing FDA-approved Red No. 3

 

 

Sodium Benzoate: A food preservative

Found in:

Fruit juice, carbonated beverages, and pickles

Sodium benzoate is found in abundance in acidic foods. It is used to retard the growth of microorganisms, according to the Center for Science in the Public Interest (CSPI).

 

Examples of foods containing FDA-approved sodium benzoate
Examples of foods containing FDA-approved sodium benzoate

 

 

Red No. 40: A food coloring (AKA Allura Red)

This food dye is the most widely used food dye in the US, exceeding both Yellow No. 5 and Yellow No. 6.

Found in:

Some Frito-Lay products; some Yoplait products; JELL-O Gelatin desserts; Quaker Instant Oatmeal; Trix; Froot-Loops; Apple Jacks; some Pop-Tart products; Kid Cuisine Kung Fu Panda products; Oscar Mayer Lunchables products; Hostess Twinkies; some Pillsbury rolls and frostings; some Betty Crocker and Duncan Hines frostings; and more

 

Some food products containing FDA-approved food dye Red No. 40
Some food products containing FDA-approved food dye Red No. 40

 

 

 

Yellow No.5: A food coloring (AKA Tartrazine)

Yellow No. 5 is the only food dye that has been tested by itself, not as part of a mix of food dyes. These studies found a link to hyperactivity.

It is the second most commonly used food dye in the US.

Found in:

Nabisco Cheese Nips Four Cheese; Frito-Lay Sun Chips Harvest Cheddar and other Frito-Lay products; some Hunt’s Snack Pack Pudding products; Lucky Charms; Eggo waffles and other waffle products; some Pop-Tarts products; various Kraft macaroni and cheese products; Betty Crocker Hamburger Helper and other products

 

Some food products containing FDA-approved food dye Yellow No. 5
Some food products containing FDA-approved food dye Yellow No. 5

Yellow No.6: A food coloring (AKA Sunset Yellow)

This is the third most widely used food dye in the US.

Found in:

Frito-Lay Cheetos Flamin’ Hot Crunchy and other Frito-Lay products; Betty Crocker Fruit Roll-ups; some JELL-O gelatin deserts and instant puddings; Fruity Cheerios; Trix; some Eggo waffle products; some Kid Cuisine Kung Fu Panda products; some Kraft macaroni and cheese dinners; some Betty Crocker frostings; some M&M’s and Skittles candies; Sunkist Orange Soda; Fanta Orange

 

 

Examples of food products containing FDA-approved food dye Yellow No. 6
Examples of food products containing FDA-approved food dye Yellow No. 6

 

 

 

COMPREHENSIVE LIST OF FDA-APPROVED DYES USED IN FOOD PRODUCTS

 

box_grey_logo

 

The Food and Health program at the Institute for Agriculture and Trade Policy (IATP) has made an extensive investigation into what’s in our food.
You can use their comprehensive IATP Brain Food Selector: A Listing of Foods and the Food Coloring they Contain to find which artificial dyes are in your child’s (and your) favorite foods.
The chart is prefaced by this information:

Synthetic food dyes, made from petrochemicals, are common in manufactured foods that are widely consumed by children. Strong science implicates food dyes with increased hyperactivity in children. That research led the British government to ask companies to stop using most dyes by December 2009. And beginning in July 2010 most dyed foods marketed throughout the European Union are required to bear a warning notice. The Center for Science in the Public Interest (CSIP) has petitioned the U.S. Food and Drug Administration to ban most food dyes in the United States. More recently, CSPI published a report, Rainbow of Risks, that discusses risks of cancer, genetic damage, and allergic reactions due to dyes.

Use IATP’s Brain Food Selector to find the dyes in your child’s (and your) favorite foods.

Note that Citrus Red 2 is approved only for use on the skins of oranges not used in processing, but consumers are almost never told of its presence. Orange B is permitted on sausage casings, but has not been used for years.

images

Also see the IATP’s Food and Health Program’s Smart Guide to Food Dyes: Buying Foods That Can Help Learning for information on health concerns posed by artificial food dyes for children and what to buy instead.
If you’re looking for citations of scientific studies, be sure to see Appendix One  on page 4 of the Smart Guide to Food Dyes (described just above) for a table of health concerns found to be caused by specific FD&C food dyes and citations of the specific research studies reporting these findings.

 

 

images-1

 

 

 

“The approach for your child’s overall health and nutrition is a diet that limits sugary and processed foods and is rich in fruits; vegetables; grains; and healthy fats, such as Omega-3 fatty acids found in fish, flaxseeds and other foods.”

– John E. Huxsahl, MD (MayoClinic.org, 2011)

 

 

 

 

(Source: thesensitivelife.weebly.com)

 

 

 

REFERENCES

AllGov.com (2014). Loopholes and Weak Enforcement Lead to Unapproved Chemicals Added to Foods. See:  http://www.allgov.com/news/controversies/loopholes-and-weak-enforcement-lead-to-unapproved-chemicals-added-to-foods-140409?news=852876

Center for Science in the Public Interest. (undated). IATP Brain Food Selector: A Listing of Foods and the Food Coloring They Contain. See:  http://brainfoodselector.iatp.org/

Curran, L. (2010).  Food Dyes Linked to Cancer, ADHD, Allergies. FOOD SAFETY NEWS. See:  http://www.foodsafetynews.com/2010/07/popular-food-dyes-linked-to-cancer-adhd-and-allergies/#.VDAh7ildXF8

Gardner, A. (2014). 9 Food Additives That May Affect ADHD. Health.com. See: http://www.health.com/health/gallery/0,,20439038_2,00.html

HealthyHolisticLiving.com. (2014). 70,000 food additives approved by the FDA – What you don’t know will hurt you. See:  http://www.healthy-holistic-living.com/70000-food-additives-approved-fda-dont-know-will-hurt.html

Huxsahl, J.E. (2011). Attention-deficit/hyperactivity disorder (ADHD) in children: What does the research say about the relationship between food additives and ADHD? MayoClinic.org. See: http://www.mayoclinic.org/diseases-conditions/adhd/expert-answers/adhd/faq-20058203

IATP’s Food and Health Program. (2009).  Smart GuideTo Food Dyes: Buying foods that can help learning. See:  http://www.iatp.org/files/421_2_105204.pdf

Kolylewski, S. & Jacobson, M. (2010). Food Dyes: A Rainbow of Risks (pdf.  See: http://cspinet.org/new/pdf/food-dyes-rainbow-of-risks.pdf

PureVolume.com. (2014). Industry Influence Found In Food Additive Reports. See:  http://www.purevolume.com/hubbardkpxf/posts/4625907/Industry+Influence+Found+In+Food+Additive+Reports

Thompson, D. (2013). Food Additive Safety Often Determined by Those With Food Industry Ties: Study. HealthDay.com. See:  http://consumer.healthday.com/public-health-information-30/ethics-health-news-747/safety-of-many-food-additives-determined-by-those-with-conflicts-of-interest-study-679010.html

 

 

 

 

DISCLAIMER:  Nothing on this site or blog is intended to provide medical advice, diagnosis or treatment.

 

© Copyright 2013-2014 Joan Rothchild Hardin. All Rights Reserved.