Tag Archives: Mood Disorders

Clue to How Gut Bacteria Affect Mood – New Evidence that Gut Bacteria Feed on a Neurotransmitter

 

 

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If you’ve read that bacteria in our guts influence our moods and have wondered how that works, here’s a new clue towards solving this piece of the recently enlivened mind/body axis puzzle.

 

 

 

THE NEUROTRANSMITTERS GABA & GLUTAMATE

 

 

(Source: www.slideshare.net)
(Source: www.slideshare.net)

 

GABA

The amino acid called GABA (Gamma Aminobutyric Acid) is the principal INHIBITORY neurotransmitter in the mammalian central nervous system,   sending chemical messages through the brain and nervous system and helping regulate communication between brain cells.
GABA’s chief role is to reduce the activity of nerve cells. It plays an important role in behavior, cognition, and  how we respond to stress. Research suggests that GABA helps control fear and anxiety when neurons become overexcited. Below normal GABA levels in the brain have been linked to depression, anxiety, sleep disorders, and schizophrenia.
Pharmaceuticals called benzodiazepines bind to the same receptors as GABA, mimicking GABA’s natural calming effects. Examples of popular benzodiazepines for anxiety and insomnia are Valium (diazepam) and Ativan (lorazepam).  They slow down the body’s central nervous system and cause sleepiness. (Konkel, 2015)
Source: www.gaba-supplement.com)
Source: www.gaba-supplement.com)

 

GLUTAMATE

Glutamate (also called L-glutamate or glutamic acid)) is another important amino acid neurotransmitter released by nerve cells in the brain. It is involved in most aspects of normal brain functioning, including cognition, memory and learning. It is the major mediator of EXCITATORY signals in the mammalian central nervous system. (Danbolt, 2001)

 

(Source: neuroplastix.com)
(Source: neuroplastix.com)

 

 

 

 

GABA & GLUTAMATE IN BALANCE

 

(Source: www.scilogs.com)
(Source: www.scilogs.com)

 

Calming GAMBA restrains the release of excitatory glutamate. So you can see that a balance between GABA and glutamate production is needed for proper functioning.  It’s a Goldilocks situation: The brain needs to release just the right amount of both GABA and glutamate. Too much or too little of one or the other causes problems.

 

 

(Source: neuroplastix.com)
(Source: neuroplastix.com)

 

 

 

 

IT TURNS OUT THAT A TYPE OF BACTERIA IN THE GUT LIVES ON GABA

Researchers have now observed gut bacteria consuming the brain chemical GABA. They found that a type of recently discovered gut bacteria, called KLE1738, can survive and reproduce only if it has GABA molecules to feed on. The researchers tried providing KLE1738 with other types of neurotransmitters but the bacteria couldn’t survive on anything but GABA. Without GABA, these bacteria die.
This is an important clue about how our gut bacteria influence our mood. “GABA acts by inhibiting signals from nerve cells, calming down the activity of the brain, so it’s surprising to learn that a gut bacterium needs it to grow and reproduce. Having abnormally low levels of GABA is linked to depression and mood disorders, and this finding adds to growing evidence that our gut bacteria may affect our brains.” (Coghlan, 2016)
An earlier experiment, in 2011, demonstrated that a different type of gut bacteria, Lactobacillus rhamnosus, dramatically altered GABA activity in the brains of mice as well as affected how well they responded to stress.
When the researchers surgically removed the vagus nerve, the communication pathway between the gut and the brain, the effect on the mice disappeared – more evidence on how gut bacteria influence the brain. (Coghlan, 2016)
The research team, led by Philip Strandwitz at Northeastern University in Boston, is now searching for other gut bacteria that consume or even produce GABA. They plan to test their effect on the brains and behavior of animals. Such work may eventually lead to new treatments for mood disorders like depression or anxiety.
“Due to this unique growth requirement, we provisionally name KLE1738 Evtepia gabavorous. Using growth of E. gabalyticus as an indicator, we then identified novel GABA producing bacteria from the gut microbiome. Reduced levels of GABA are associated with depression, and we found fewer GABA producers in a human cohort of depressed individuals. By modulating the level of GABA, microbial producers and consumers of this neurotransmitter may be influencing host behavior.” (Strandwitz et al, 2016)

 

 

(Source: www.slideshare.net)
(Source: www.slideshare.net)

 

 

Researchers are just at the beginning of looking into the many ways the gut microbiome influences, if not regulates, many bodily processes and how unbalance in the gut microbiome eventually leads to poor health.

 

 

(Source: www.medicaldaily.com)
(Source: www.medicaldaily.com)

 

This finding of a dependence of a type of gut bacteria on the neurotransmitter GABA doesn’t mean you should start yourself on one of the GABA supplements you’ll find for sale online. But do stay tuned! Neurotransmitters and specific microbes may become the treatment of choice for mood disorders – or, even better, for preventing mood disorders in the first place.
Keep your gut microbiome health, keep your body healthy.

 

 

(Source: slideplayer.com)
(Source: slideplayer.com)

 

 

 

REFERENCES

Coghlan, A. (2016). Gut bacteria spotted eating brain chemicals for the first time. NewScientist. com. See: https://www.newscientist.com/article/2095769-gut-bacteria-spotted-eating-brain-chemicals-for-the-first-time/

Danbolt, N.C. (2001). Glutamate as a Neurotransmitter – An overview. Center for Molecular Biology & Neuroscience, The Neurotransporter Group – Dynamics of extracellular transmitter amino acids. See: http://neurotransporter.org/glutamate.html

Konkel, G. (2015). What Is GABA? See: http://www.everydayhealth.com/gaba/guide/

Strandwitz et al. (2016). Gaba Modulating Bacteria of the Human Gut Microbiome.  American Society for Microbiology 2016, Session 347 – Microbial Mind Control. See: http://www.abstractsonline.com/pp8/#!/4060/presentation/18619

 

 

 

© Copyright 2016. Joan Rothchild Hardin. All Rights Reserved.

 

DISCLAIMER:  Nothing on this site or blog is intended to provide medical advice, diagnosis or treatment.

 

How Sugar Affects Your Health – 146 Ways

 

 

(Source: glutenfabulous.org)
(Source: glutenfabulous.org)

 

This list of 146 way sugar affects our health – all detrimental – was compiled by Nancy Appleton, PhD from medical journals and other scientific publications. Dr Appleton is a clinical nutritionist and researcher. She is the author of several books, including Lick The Sugar Habit, Stopping Inflammation: Relieving the Cause of Degenerative Diseases, and Suicide by Sugar: A Startling Look at Our #1 National Addiction. Her website is www.nancyappleton.com

 

1. Sugar can suppress the immune system.

2. Sugar upsets the mineral relationships in the body.

3. Sugar can cause hyperactivity, anxiety, difficulty concentrating, and crankiness in children.

4. Sugar can produce a significant rise in triglycerides.

5. Sugar contributes to the reduction in defense against bacterial infection (infectious diseases).

6. Sugar causes a loss of tissue elasticity and function, the more sugar you eat the more elasticity and function you loose.

7. Sugar reduces high density lipoproteins.

8. Sugar leads to chromium deficiency.

9 Sugar leads to cancer of the ovaries.

10. Sugar can increase fasting levels of glucose.

11. Sugar causes copper deficiency.

12. Sugar interferes with absorption of calcium and magnesium.

13. Sugar can weaken eyesight.

14. Sugar raises the level of a neurotransmitters: dopamine, serotonin, and norepinephrine.

15. Sugar can cause hypoglycemia.

16. Sugar can produce an acidic digestive tract.

17. Sugar can cause a rapid rise of adrenaline levels in children.

18. Sugar malabsorption is frequent in patients with functional bowel disease.

19. Sugar can cause premature aging.

20. Sugar can lead to alcoholism.

21. Sugar can cause tooth decay.

22. Sugar contributes to obesity

23. High intake of sugar increases the risk of Crohn’s disease and ulcerative colitis.

24. Sugar can cause changes frequently found in person with gastric or duodenal ulcers.

25. Sugar can cause arthritis.

26. Sugar can cause asthma.

27. Sugar greatly assists the uncontrolled growth of Candida Albicans (yeast infections).

28. Sugar can cause gallstones.

29. Sugar can cause heart disease.

30. Sugar can cause appendicitis.

31. Sugar can cause multiple sclerosis.

32. Sugar can cause hemorrhoids.

33. Sugar can cause varicose veins.

34. Sugar can elevate glucose and insulin responses in oral contraceptive users.

35. Sugar can lead to periodontal disease.

36. Sugar can contribute to osteoporosis.

37. Sugar contributes to saliva acidity.

38. Sugar can cause a decrease in insulin sensitivity.

39. Sugar can lower the amount of Vitamin E (alpha-Tocopherol in the blood.

40. Sugar can decrease growth hormone.

41. Sugar can increase cholesterol.

42. Sugar can increase the systolic blood pressure.

43. Sugar can cause drowsiness and decreased activity in children.

44. High sugar intake increases advanced glycation end products (AGEs)(Sugar bound non-enzymatically to protein)

45. Sugar can interfere with the absorption of protein.

46. Sugar causes food allergies.

47. Sugar can contribute to diabetes.

48. Sugar can cause toxemia during pregnancy.

49. Sugar can contribute to eczema in children.

50. Sugar can cause cardiovascular disease.

51. Sugar can impair the structure of DNA

52. Sugar can change the structure of protein.

53. Sugar can make our skin age by changing the structure of collagen.

54. Sugar can cause cataracts.

55. Sugar can cause emphysema.

56. Sugar can cause atherosclerosis.

57. Sugar can promote an elevation of low density lipoproteins (LDL).

58. High sugar intake can impair the physiological homeostasis of many systems in the body.

59. Sugar lowers the enzymes ability to function.

60. Sugar intake is higher in people with Parkinson’s disease.

61. Sugar can cause a permanent altering the way the proteins act in the body.

62. Sugar can increase the size of the liver by making the liver cells divide.

63. Sugar can increase the amount of liver fat.

64. Sugar can increase kidney size and produce pathological changes in the kidney.

65. Sugar can damage the pancreas.

66. Sugar can increase the body’s fluid retention.

67. Sugar is enemy #1 of the bowel movement.

68. Sugar can cause myopia (nearsightedness).

69. Sugar can compromise the lining of the capillaries.

70. Sugar can make the tendons more brittle.

71. Sugar can cause headaches, including migraine.

72. Sugar plays a role in pancreatic cancer in women.

73. Sugar can adversely affect school children’s grades and cause learning disorders..

74. Sugar can cause an increase in delta, alpha, and theta brain waves.

75. Sugar can cause depression.

76. Sugar increases the risk of gastric cancer.

77. Sugar and cause dyspepsia (indigestion).

78. Sugar can increase your risk of getting gout.

79. Sugar can increase the levels of glucose in an oral glucose tolerance test over the ingestion of complex carbohydrates.

80. Sugar can increase the insulin responses in humans consuming high-sugar diets compared to low sugar diets.

81 High refined sugar diet reduces learning capacity.

82. Sugar can cause less effective functioning of two blood proteins, albumin, and lipoproteins, which may reduce the body’s ability to handle fat and cholesterol.

83. Sugar can contribute to Alzheimer’s disease.

84. Sugar can cause platelet adhesiveness.

85. Sugar can cause hormonal imbalance; some hormones become underactive and others become overactive.

86. Sugar can lead to the formation of kidney stones.

87. Sugar can lead to the hypothalamus to become highly sensitive to a large variety of stimuli.

88. Sugar can lead to dizziness.

89. Diets high in sugar can cause free radicals and oxidative stress.

90. High sucrose diets of subjects with peripheral vascular disease significantly increases platelet adhesion.

91. High sugar diet can lead to biliary tract cancer.

92. Sugar feeds cancer.

93. High sugar consumption of pregnant adolescents is associated with a twofold increased risk for delivering a small-for-gestational-age (SGA) infant.

94. High sugar consumption can lead to substantial decrease in gestation duration among adolescents.

95. Sugar slows food’s travel time through the gastrointestinal tract.

96. Sugar increases the concentration of bile acids in stools and bacterial enzymes in the colon. This can modify bile to produce cancer-causing compounds and colon cancer.

97. Sugar increases estradiol (the most potent form of naturally occurring estrogen) in men.

98. Sugar combines and destroys phosphatase, an enzyme, which makes the process of digestion more difficult.

99. Sugar can be a risk factor of gallbladder cancer.

100. Sugar is an addictive substance.

101. Sugar can be intoxicating, similar to alcohol.

102. Sugar can exacerbate PMS.

103. Sugar given to premature babies can affect the amount of carbon dioxide they produce.

104. Decrease in sugar intake can increase emotional stability.

105. The body changes sugar into 2 to 5 times more fat in the bloodstream than it does starch.

106. The rapid absorption of sugar promotes excessive food intake in obese subjects.

107. Sugar can worsen the symptoms of children with attention deficit hyperactivity disorder (ADHD).

108. Sugar adversely affects urinary electrolyte composition.

109. Sugar can slow down the ability of the adrenal glands to function.

110. Sugar has the potential of inducing abnormal metabolic processes in a normal healthy individual and to promote chronic degenerative diseases.

111.. IVs (intravenous feedings) of sugar water can cut off oxygen to the brain.

112. High sucrose intake could be an important risk factor in lung cancer.

113. Sugar increases the risk of polio.

114. High sugar intake can cause epileptic seizures.

115. Sugar causes high blood pressure in obese people.

116. In Intensive Care Units, limiting sugar saves lives.

117. Sugar may induce cell death.

118. Sugar can increase the amount of food that you eat.

119. In juvenile rehabilitation camps, when children were put on a low sugar diet, there was a 44% drop in antisocial behavior.

120. Sugar can lead to prostate cancer.

121. Sugar dehydrates newborns.

122. Sugar increases the estradiol in young men.

123. Sugar can cause low birth weight babies.

124. Greater consumption of refined sugar is associated with a worse outcome of schizophrenia

125. Sugar can raise homocysteine levels in the blood stream.

126. Sweet food items increase the risk of breast cancer.

127. Sugar is a risk factor in cancer of the small intestine.

128. Sugar may cause laryngeal cancer.

129. Sugar induces salt and water retention.

130. Sugar may contribute to mild memory loss.

131. As sugar increases in the diet of 10 years olds, there is a linear decrease in the intake of many essential nutrients.

132. Sugar can increase the total amount of food consumed.

133. Exposing a newborn to sugar results in a heightened preference for sucrose relative to water at 6 months and 2 years of age.

134. Sugar causes constipation.

135. Sugar causes varicose veins.

136. Sugar can cause brain decay in prediabetic and diabetic women.

137. Sugar can increase the risk of stomach cancer.

138. Sugar can cause metabolic syndrome.

139. Sugar ingestion by pregnant women increases neural tube defects in embryos.

140. Sugar can be a factor in asthma.

141. The higher the sugar consumption the more chances of getting irritable bowel syndrome.

142. Sugar could affect central reward systems.

143. Sugar can cause cancer of the rectum.

144. Sugar can cause endometrial cancer.

145. Sugar can cause renal (kidney) cell carcinoma.

146. Sugar can cause liver tumors.

 

 

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Many thanks to Dr Beth Forgosh, of Discover Chiropractic of Soho, for bringing Dr Appleton’s list to my attention.

 

 

Note added to this post on 12/29/2014:

 

fruit-vs-dessert

 

Suzette Lawrence, MSN, commented that Dr Appleton’s list, above, describes the effects of REFINED sugars:

“This is not the case for natural fruits sugars that are attached to the fiber in the fruit, known as levulose … if absorbed it occurs low in the intestines and is converted to glycogen in the liver and stored there as an emergency energy source.  I agree that the SAD (Standard American Diet) beginning in infancy sets the stage for every disease, and some new ones. Think, GMO beet sugar … ”

From a 2014 article by the Cancer Treatment Centers of America entitled Natural vs. refined sugars – What’s the difference?:

Sugar, in all forms, is a simple carbohydrate that the body converts into glucose and uses for energy. But the effect on the body and your overall health depends on the type of sugar you’re eating, either natural or refined.

We wanted to explore the difference between these sugar types as a follow-up to our post about whether sugar drives the growth of cancer, which has received several comments. We again turned to Julie Baker, Clinical Oncology Dietitian at our hospital outside Atlanta, for her expertise on the issue.

Understanding sugars

Natural sugars are found in fruit as fructose and in dairy products, such as milk and cheese, as lactose. Foods with natural sugar have an important role in the diet of cancer patients and anyone trying to prevent cancer because they provide essential nutrients that keep the body healthy and help prevent disease.

Refined sugar comes from sugar cane or sugar beets, which are processed to extract the sugar. It is typically found as sucrose, which is the combination of glucose and fructose. We use white and brown sugars to sweeten cakes and cookies, coffee, cereal and even fruit. Food manufacturers add chemically produced sugar, typically high-fructose corn syrup, to foods and beverages, including crackers, flavored yogurt, tomato sauce and salad dressing. Low-fat foods are the worst offenders, as manufacturers use sugar to add flavor.

Most of the processed foods we eat add calories and sugar with little nutritional value. In contrast, fruit and unsweetened milk have vitamins and minerals. Milk also has protein and fruit has fiber, both of which keep you feeling full longer.

DR APPLETON’S REFERENCES

1. Sanchez, A., et al. Role of Sugars in Human Neutrophilic Phagocytosis, American Journal of Clinical Nutrition. Nov 1973;261:1180-1184.
Bernstein, J., et al. Depression of Lymphocyte Transformation Following Oral Glucose Ingestion. American Journal of Clinical Nutrition.1997;30:613.
2. Couzy, F., et al. Nutritional Implications of the Interaction Minerals, Progressive Food and Nutrition Science 17;1933:65-87.
3. Goldman, J., et al. Behavioral Effects of Sucrose on Preschool Children. Journal of Abnormal Child Psychology. 1986;14(4):565-577.
4. Scanto, S. and Yudkin, J. The Effect of Dietary Sucrose on Blood Lipids, Serum Insulin, Platelet Adhesiveness and Body Weight in Human Volunteers, Postgraduate Medicine Journal. 1969;45:602-607.
5. Ringsdorf, W., Cheraskin, E. & Ramsay R. Sucrose,Neutrophilic Phagocytosis and Resistance to Disease, Dental Survey. 1976;52(12):46-48.
6. Cerami, A., Vlassara, H., & Brownlee, M. Glucose and Aging. Scientific American. May 1987:90.
Lee, A. T. & Cerami, A. The Role of Glycation in Aging. Annals of the New York Academy of Science. 663:63-67.
7. Albrink, M. & Ullrich I. H. Interaction of Dietary Sucrose and Fiber on Serum Lipids in Healthy Young Men Fed High Carbohydrate Diets. American Journal of Clinical Nutrition. 1986;43:419-428.
Pamplona, R., et al. Mechanisms of Glycation in Atherogenesis. Medical Hypotheses. Mar 1993;40(3):174-81.
8. Kozlovsky, A., et al. Effects of Diets High in Simple Sugars on Urinary Chromium Losses. Metabolism. June 1986;35:515-518.
9. Takahashi, E., Tohoku University School of Medicine, Holistic Health Digest. October 1982:41.
10. Kelsay, J., et al. Diets High in Glucose or Sucrose and Young Women. American Journal of Clinical Nutrition. 1974;27:926-936.
Thomas, B. J., et al. Relation of Habitual Diet to Fasting Plasma Insulin Concentration and the Insulin Response to Oral Glucose. Human Nutrition Clinical Nutrition. 1983; 36C(1):49_51.
11. Fields, M., et al. Effect of Copper Deficiency on Metabolism and Mortality in Rats Fed Sucrose or Starch Diets, Journal of Clinical Nutrition. 1983;113:1335-1345.
12. Lemann, J. Evidence that Glucose Ingestion Inhibits Net Renal Tubular Reabsorption of Calcium and Magnesium. Journal Of Clinical Nutrition. 1976 ;70:236-245.
13. Acta Ophthalmologica Scandinavica. Mar 2002;48;25.
Taub, H. Ed. Sugar Weakens Eyesight, VM NEWSLETTER; May 1986:6
14. Sugar, White Flour Withdrawal Produces Chemical Response. The Addiction Letter. Jul 1992:4.
15. Dufty, William. Sugar Blues. (New York:Warner Books, 1975).
16. Ibid.
17. Jones, T. W., et al. Enhanced Adrenomedullary Response and Increased Susceptibility to Neuroglygopenia: Mechanisms Underlying the Adverse Effect of Sugar Ingestion in Children. Journal of Pediatrics. Feb 1995;126:171-7.
18. Ibid.
19. Lee, A. T. & Cerami A. The Role of Glycation in Aging.” Annals of the New York Academy of Science.1992;663:63-70.
20. Abrahamson, E. & Peget, A. Body, Mind and Sugar. (New York:Avon,1977.}
21. Glinsmann, W., Irausquin, H., & Youngmee, K. Evaluation of Health Aspects of Sugar Contained in Carbohydrate Sweeteners. F. D. A. Report of Sugars Task Force. 1986:39.
Makinen K.K.,et al. A Descriptive Report of the Effects of a 16-month Xylitol Chewing-Gum Programme Subsequent to a 40-Month Sucrose Gum Programme. Caries Research. 1998; 32(2)107-12.
Riva Touger-Decker & Cor van Loveren, Sugars and Dental Caries.
American Journal of Clinical Nutrition. Oct 2003; 78:881-892.
22. Keen, H., et al. Nutrient Intake, Adiposity, and Diabetes. British Medical Journal. 1989; 1: 655-658.
23. Tragnone, A. et al. Dietary Habits as Risk Factors for Inflammatory Bowel Disease. European Journal of Gastroenterological Hepatology. Jan 1995;7(1):47-51.
24. Yudkin, J. Sweet and Dangerous. (New York;Bantam Books:1974), 129.
25. Darlington, L., Ramsey, N. W. & Mansfield, J. R. Placebo_Controlled, Blind Study of Dietary Manipulation Therapy in Rheumatoid Arthritis, Lancet. Feb 1986;8475(1):236-238.
26. Powers, L. Sensitivity: You React to What You Eat. Los Angeles Times. Feb. 12, 1985.
Cheng, J., et al. Preliminary Clinical Study on the Correlation Between Allergic Rhinitis and Food Factors. Lin Chuang Er Bi Yan Hou Ke Za Zhi Aug 2002;16(8):393-396.
27. Crook, W. J. The Yeast Connection. (TN:Professional Books, 1984)..
28. Heaton, K. The Sweet Road to Gallstones. British Medical Journal. Apr 14, 1984; 288:1103-1104.
Misciagna, G., et al. American Journal of Clinical Nutrition. 1999;69:120-126.
29. Yudkin, J. Sugar Consumption and Myocardial Infarction. Lancet.Feb 6, 1971;1(7693):296-297.
Reiser, S. Effects of Dietary Sugars on Metabolic Risk Factors Associated with Heart Disease. Nutritional Health. 1985;203-216.
30. Cleave, T. The Saccharine Disease. (New Canaan, CT: Keats Publishing, 1974).
31. Erlander, S. The Cause and Cure of Multiple Sclerosis, The Disease to End Disease. Mar 3, 1979;1(3):59-63.
32. Cleave, T. The Saccharine Disease. (New Canaan, CT: Keats Publishing, 1974.)
33. Cleave, T. & Campbell, G. Diabetes, Coronary Thrombosis and the Saccharine Disease. (Bristol, England, John Wrightand Sons, 1960).
34. Behall, K. Influence of Estrogen Content of Oral Contraceptives and Consumption of Sucrose on Blood Parameters. Disease Abstracts International. 1982;431-437.
35. Glinsmann, W., Irausquin, H., & K. Youngmee. Evaluation of Health Aspects of Sugar Contained in Carbohydrate Sweeteners. F. D. A. Report of Sugars Task Force. 1986;39:36_38.
36. Tjderhane, L. & Larmas, M. A High Sucrose Diet Decreases the Mechanical Strength of Bones in Growing Rats. Journal of Nutrition. 1998:128:1807-1810.
37. Appleton, N. Healthy Bones. New York: Avery Penguin Putnam,1989.
38. Beck_Nielsen H., Pedersen O., & Schwartz S. Effects of Diet on the Cellular Insulin Binding and the Insulin Sensitivity in Young Healthy Subjects. Diabetes. 1978;15:289-296 .
39. Mohanty P. et al. Glucose Challenge Stimulates Reactive Oxygen Species (ROS) Generation by Leucocytes. Journal of Clinical Endocrinology and Metabolism. Aug 2000; 85(8):2970-2973.
40. Gardner, L. & Reiser, S. Effects of Dietary Carbohydrate on Fasting Levels of Human Growth Hormone and Cortisol. Proceedings of the Society for Experimental Biology and Medicine. 1982;169:36-40.
41. Reiser, S. Effects of Dietary Sugars on Metabolic Risk Factors Associated with Heart Disease. Nutritional Health. 1985;203:216.
42. Preuss, H. G. Sugar-Induced Blood Pressure Elevations Over the Lifespan of Three Substrains of Wistar Rats. Journal of the American College of Nutrition, 1998;17(1) 36-37.
43. Behar, D., et al. Sugar Challenge Testing with Children Considered Behaviorally Sugar Reactive. Nutritional Behavior. 1984;1:277-288.
44. Furth, A. & Harding, J. Why Sugar Is Bad For You. New Scientist. Sep 23, 1989;44.
45. Lee AT, & Cerami A. Role of Glycation in Aging. Annals of the New York Academy of Science. Nov 21,1992 ;663:63-70.
46. Appleton, N. Lick the Sugar Habit. (New York:Avery Penguin Putnam:1988).
47. Sucrose Induces Diabetes in Cats. Federal Protocol. 1974;6(97).
48. Cleave, T. The Saccharine Disease (New Canaan Ct: Keats Publishing, Inc., 1974).131.
49. Ibid. 132.
50. Vaccaro O., Ruth, K. J. & Stamler J. Relationship of Postload Plasma Glucose to Mortality with 19 Year Follow-up. Diabetes Care. Oct 15,1992;10:328-334.
Tominaga, M., et al, Impaired Glucose Tolerance Is a Risk Factor for Cardiovascular Disease, but Not Fasting Glucose. Diabetes Care. 1999:2(6):920-924.
51. Lee, A. T. & Cerami, A. Modifications of Proteins and Nucleic Acids by Reducing Sugars: Possible Role in Aging. Handbook of the Biology of Aging. (New York: Academic Press, 1990.).
52. Monnier, V. M. Nonenzymatic Glycosylation, the Maillard Reaction and the Aging Process. Journal of Gerontology. 1990:45(4 ):105-110.
53. Dyer, D. G., et al. “=Accumulation of Maillard Reaction Products in Skin Collagen in Diabetes and Aging. Journal of Clinical Investigation. 1993:93(6):421-422.
54. Veromann, S.et al. Dietary Sugar and Salt Represent Real Risk Factors for Cataract Development. Ophthalmologica. Jul-Aug 2003 ;217(4):302-307.
55. Monnier, V. M. Nonenzymatic Glycosylation, the Maillard Reaction and the Aging Process. Journal of Gerontology. 1990:45(4):105-110.
56. Schmidt A.M. et al. Activation of receptor for advanced glycation end products: a mechanism for chronic vascular dysfunction in diabetic vasculopathy and atherosclerosis. Circular Research Archives. 1999 Mar 19;84(5):489-97.
57. Lewis, G. F. and Steiner, G. Acute Effects of Insulin in the Control of VLDL Production in Humans. Implications for Theinsulin-resistant State. Diabetes Care. 1996 Apr;19(4):390-3
R. Pamplona, M. .J., et al. Mechanisms of Glycation in Atherogenesis. Medical Hypotheses. 1990;40:174-181.
58. Ceriello, A. Oxidative Stress and Glycemic Regulation. Metabolism. Feb 2000;49(2 Suppl 1):27-29.
59. Appleton, Nancy. Lick the Sugar Habit. (New York:Avery Penguin Putnam, 1988).
60. Hellenbrand, W. Diet and Parkinson’s Disease. A Possible Role for the Past Intake of Specific Nutrients. Results from a Self-administered Food-frequency Questionnaire in a Case-control Study. Neurology. Sep 1996;47(3):644-650 Cerami, A., Vlassara, H., & Brownlee, M. Glucose and Aging. Scientific American. May 1987: 90.
62. Goulart, F. S. Are You Sugar Smart? American Fitness. Mar-Apr 1991: 34-38.
63. Ibid.
64. Yudkin, J., Kang, S. & Bruckdorfer, K. Effects of High Dietary Sugar. British Journal of Medicine. Nov 22, 1980;1396.
65. Goulart, F. S. Are You Sugar Smart? American Fitness. March_April 1991: 34-38
66. Ibid.
67. Ibid.
68. Ibid.
69. Ibid.
70. Nash, J. Health Contenders. Essence. Jan 1992-23: 79_81.
71. Grand, E. Food Allergies and Migraine. Lancet. 1979:1:955_959.
72. Michaud, D. Dietary Sugar, Glycemic Load, and Pancreatic Cancer Risk in a Prospective Study. Journal of the National Cancer Institute. Sep 4, 2002 ;94(17):1293-300.
73. Schauss, A. Diet, Crime and Delinquency. (Berkley Ca; Parker House, 1981).
74. Christensen, L. The Role of Caffeine and Sugar in Depression. Nutrition Report. Mar 1991;9(3):17-24.
75. Ibid.
76. Cornee, J., et al. A Case-control Study of Gastric Cancer and Nutritional Factors in Marseille, France, European Journal of Epidemiology. 1995;11:55-65.
77. Yudkin, J. Sweet and Dangerous.(New York:Bantam Books,1974) 129.
78. Ibid, 44
79. Reiser, S., et al. Effects of Sugars on Indices on Glucose Tolerance in Humans. American Journal of Clinical Nutrition. 1986:43;151-159.
80. Reiser,S., et al. Effects of Sugars on Indices on Glucose Tolerance in Humans.  American Journal of Clinical Nutrition. 1986;43:151-159.
81. Molteni, R, et al. A High-fat, Refined Sugar Diet Reduces Hippocampal Brain-derived Neurotrophic Factor, Neuronal Plasticity, and Learning. NeuroScience. 2002;112(4):803-814.
82. Monnier, V., Nonenzymatic Glycosylation, the Maillard Reaction and the Aging Process. Journal of Gerontology. 1990;45:105-111.
83. Frey, J. Is There Sugar in the Alzheimers Disease? Annales De Biologie Clinique. 2001; 59 (3):253-257.
84. Yudkin, J. Metabolic Changes Induced by Sugar in Relation to Coronary Heart Disease and Diabetes. Nutrition and Health. 1987;5(1-2):5-8.
85. Ibid.
86. Blacklock, N. J., Sucrose and Idiopathic Renal Stone. Nutrition and Health. 1987;5(1-2):9-12.
Curhan, G., et al. Beverage Use and Risk for Kidney Stones in Women. Annals of Internal Medicine. 1998:28:534-340.
87. Journal of Advanced Medicine. 1994;7(1):51-58.
88. Ibid.
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99. Moerman, C. et al. Dietary Sugar Intake in the Etiology of Gallbladder Tract Cancer. International Journal of Epidemiology. Apr 1993; 22(2):207-214.
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103. Sunehag, A. L., et al. Gluconeogenesis in Very Low Birth Weight Infants Receiving Total Parenteral Nutrition. Diabetes. 1999 ;48 7991-8000).
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105. Nutrition Health Review. Fall 85. Sugar Changes into Fat Faster than Fat.
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107. Girardi, N.L. Blunted Catecholamine Responses after Glucose Ingestion in Children with Attention Deficit Disorder. Pediatrics Research. 1995;38:539-542.
Berdonces, J. L. Attention Deficit and Infantile Hyperactivity. Rev Enferm. Jan 2001;4(1)11-4
108. Blacklock, N. J. Sucrose and Idiopathic Renal Stone. Nutrition Health. 1987;5(1 & 2):9-17.
109. Lechin, F., et al. Effects of an Oral Glucose Load on Plasma Neurotransmitters in Humans. Neurophychobiology. 1992;26(1-2):4-11.
110. Fields, M. Journal of the American College of Nutrition. Aug 1998;17(4):317-321.
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113. Sandler, Benjamin P. Diet Prevents Polio. Milwakuee, WI,:The Lee Foundation for for Nutritional Research, 1951.
114. Murphy, Patricia. The Role of Sugar in Epileptic Seizures. Townsend Letter for Doctors and Patients. May, 2001.
115. Stern, N. & Tuck, M. Pathogenesis of Hypertension in Diabetes Mellitus. Diabetes Mellitus, a Fundamental and Clinical Test. 2nd Edition, (Phil. A: Lippincott Williams & Wilkins, 2000)943-957.
116. Christansen, D. Critical Care: Sugar Limit Saves Lives. Science News. June 30, 2001;159:404.
117. Donnini, D. et al. Glucose May Induce Cell Death through a Free Radical-mediated Mechanism.Biochem Biohhys Res Commun. Feb 15, 1996:219(2):412-417.
118. Allen S. Levine, Catherine M. Kotz, & Blake A. Gosnell . Sugars and Fats: The Neurobiology of Preference. Journal of Nutrition. 2003 133:831S-834S.
119. Schoenthaler, S. The Los Angeles Probation Department Diet-Behavior Program: An Empirical Analysis of Six Institutional Settings. International Journal of Biosocial Research. 5(2):88-89.
120. Deneo-Pellegrini H,. et al. Foods, Nutrients and Prostate cancer: a Case-control study in Uruguay. Br J Cancer. 1999 May;80(3-4):591-7.
121. Gluconeogenesis in Very Low Birth Weight Infants Receiving Total Parenteral Nutrition. Diabetes. 1999 Apr;48(4):791-800.
122. Yudkin, J. and Eisa, O. Dietary Sucrose and Oestradiol Concentration in Young Men. Annals of Nutrition and Metabolism. 1988;32(2):53-5.
123. Lenders, C. M. Gestational Age and Infant Size at Birth Are Associated with Dietary Intake Among Pregnant Adolescents. Journal of Nutrition.128; 1998::807-1810.
124. Peet, M. International Variations in the Outcome of Schizophrenia and the Prevalence of Depression in Relation to National Dietary Practices: An Ecological
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125. Fonseca, V. et al. Effects of a High-fat-sucrose Diet on Enzymes in Homosysteine Metabolism in the Rat. Metabolism. 200; 49:736-41.
126. Potischman, N, et.al. Increased Risk of Early-stage Breast Cancer Related to Consumption of Sweet Foods among Women Less than Age 45 in the United States. Cancer Causes Control. 2002 Dec;13(10):937-46.
127.Negri. E. et al. Risk Factors for Adenocarcinoma of the Small Intestine.
International Journal of Cancer. 1999:82:I2:171-174.
128.Bosetti, C. et al. Food Groups and Laryngeal Cancer Risk: A Case-control Study from Italy and Switzerland. International Journal of Cancer, 2002:100(3): 355-358.
129. Shannon, M. An Empathetic Look at Overweight.CCL Family Foundation. Nov-Dec.1993. 20(3):3-5.
130. Harry G. Preuss, MD, of Georgetown University Medical School.
131. Health After 50. Johns Hopkins Medical Letter. May, 1994.
132. Allen, S. Sugars and Fats: The Neurobiology of Preference. Journal of Nutrition. 2003;133:831S-834S.
133. Booth, D.A.M. et al. Sweetness and Food Selection: Measurement of Sweeteners Effects on Acceptance. Sweetness. Dobbing, J., Ed., (London:Springer-Verlag, 1987).
134. Cleve, T.L On the Causation of Varicose Veins. Bristol, England, John Wright, 1960.
135. Cleve, T.L On the Causation of Varicose Veins. Bristol, England, John Wright, 1960.
136. Ket, Yaffe et al. Diabetes, Impaired Fasting Glucose and Development of Cognitive Impairment in Older Women. Neurology. 2004;63:658�663.
137. Chatenoud, Liliane et al. Refined-cereal Intake and Risk of Selected Cancers in Italy. American Journal of Clinical Nutrition, Dec 1999;70:1107-1110.
138. Yoo, Sunmi et al. Comparison of Dietary Intakes Associated with Metabolic Syndrome Risk Factors in Young Adults: the Bogalusa Heart Study. American Journal of Clinical Nutrition.  2004 Oct;80(4):841-848.
139. Shaw, Gary M. et al. Neural Tube Defects Associated with Maternal Periconceptional Dietary Intake of Simple Sugars and Glycemic Index.
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140. Krilanovich, Nicholas J. Fructose Misuse, the Obesity Epidemic, the Special Problems of the Child, and a Call to Action  American Journal of Clinical Nutrition, Nov 2004;80:1446-1447.
141.Jarnerot, G., Consumption of Refined Sugar by Patients with Crohn’s Disease, Ulcerative colitis, or Irritable Bowel Syndrome. Scandinavian Journal of Gastroenterology. 1983 Nov;18(8):999-1002.
142. Allen, S. Sugars and Fats: The Neurobiology of Preference. Journal of Nutrition.
2003;133:831S-834S.
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144. Levi F, Franceschi S, Negri E, & La Vecchia C. Dietary Factors and the Risk of Endometrial Cancer. Cancer. 1993 Jun 1;71(11):3575-3581.
145. Mellemgaard A. et al. Dietary Risk Factors for Renal Cell Carcinoma in Denmark. European Journal of Cancer. 1996 Apr;32A(4):673-82.
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© Copyright 2014 Joan Rothchild Hardin. All Rights Reserved.

 

DISCLAIMER:  Nothing on this site or blog is intended to provide medical advice, diagnosis or treatment.

Moms to EPA: Recall Monsanto’s Roundup

An article called MOMS TO EPA: RECALL MONSANTO’S ROUNDUP arrived in my inbox this morning, the day after I’d posted Genetically Modified Organisms – Our Food.
It tells the  stories of  how two groups of mothers, Moms Across America and Thinking Moms Revolution, discovered that the serious health problems their children suffered from were linked to exposure to the chemical glyphosate, the active ingredient in Monsanto’s Roundup.
When these moms had their sick children and the rest of their families’ glyphosate levels checked, the tests revealed high, unsafe levels in their children’s urine, in the families’ drinking water, and in the mothers’ breast milk.
This turned them into activists who are now taking action to get the U.S. Environmental Protection Agency (EPA) to recall Monsanto’s Roundup, the most widely used herbicide in the world.
The stories of their children’s suffering from entirely preventable illnesses are heart breaking – their stories of how they brought their children back to health by removing all GMOs from their diets are inspiring.
I’m also reprinting the article here because it contains links to the plentiful scientific research findings that demonstrate the serious harm being done to humans, animals and the environment by glyphosate. I’ve added a Source list after the article.
Glyphosate is the active chemical in Monsanto’s herbicide Roundup, which is sprayed on crops grown from its Roundup Ready seeds. Roundup Ready seeds have been genetically engineered to be resistant to glyphosate – allowing farmers to douse their crops with Roundup herbicide without killing the crops themselves.
Since Monsanto introduced Roundup in 1975, it has become the best-selling herbicide in the world. Its prolific use has led to the emergence of glyphosate-resistant weeds – inducing farmers to spray ever heavier amounts of Roundup on their crops.

Note: I’ve added a list of sources to the bottom of the original article.

 

 

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The Organic Consumers Association’s article:

 

Moms to EPA: Recall Monsanto’s Roundup
By Alexis Baden-Mayer
Organic Consumers Association, May 29, 2014

For related articles and information, please visit OCA’s  All About Organics page, our Genetic Engineering page, and our Millions Against Monsanto page.

 

Hell hath no fury . . . like a mother whose child has been sickened by a toxin that’s almost impossible to avoid.

Two activist groups, Moms Across America and Thinking Moms Revolution, want the U.S. Environmental Protection Agency (EPA) to recall Monsanto’s Roundup, the most widely used herbicide/pesticide in the world.

Now is the time to do it, they say, because the EPA is conducting a registration review of glyphosate.

Representatives of the two groups contacted the EPA to request a meeting. When the EPA ignored them, they rallied supporters. In just three days, about 10,000 moms from all over the country rang the phones off the hook at the EPA.

A week later, five Moms Across America leaders were sitting around a boardroom table with nine EPA employees who have the power to recall Roundup. The moms brought lawyers, scientists and advocates from Organic Consumers Association, Natural Resources Defense Council, Consumers Union, Beyond Pesticides and the Truth-In-Labeling Coalition as back-up.

What was supposed to be a one-hour meeting turned into two. The EPA’s Dana Vogel, director of the Health Effects Division in the Office of Pesticide Programs, and other EPA staff stayed glued to their seats as one mother after another shared heart-wrenching stories of parenting children with life-threatening allergies, severe gastrointestinal problems, mysterious autism-spectrum disorders, and major nutritional deficiencies.

The common thread in those stories? Exposure to glyphosate, the key ingredient in Monsanto’s Roundup.

Wrenching tales of preventable illnesses

The activist moms had long suspected pesticides might be behind their children’s health problems. So they had their families tested for glyphosate. The tests showed unsafe levels of glyphosate in their drinking water, in their breast milk and in their children’s urine.

That’s when they resolved to get in front of the EPA. And when they did, they told their stories.

Moms Across America co-founder Zen Honeycutt recounted how when she learned of the link between glyphosate and autism, she had her middle child, who had been exhibiting autism symptoms, tested for glyphosate. His urine had 8.7 parts per billion glyphosate—eight times more than is allowed in drinking water in the E.U. She immediately eliminated all potential sources of glyphosate from his diet. After six weeks, the glyphosate was out of his system. And so were the autism symptoms. He stopped hitting people, and his grades went back up from D’s to A’s.

After a year of eating organic, her eldest son’s walnut allergy went from a 19 to a 0.2. It’s no longer life-threatening.

In fact, all of the mothers’ children suffered from deteriorating conditions until they put them on all-organic diets. When they figured out that going organic was the only thing that helped ease their children’s symptoms, they started investigating the food they had been eating for possible causes of their children’s poor health.

Each mother began to suspect glyphosate.

Zoe Swartz, leader of East Coast Moms Across America and founder of GMO Free Lancaster County told the EPA, “I’m really angry that I didn’t know that there was glyphosate in the food I was feeding my daughter.” She described her toddler’s problems with “leaky gut syndrome” which has been linked to glyphosate exposure. After three weeks of an organic diet, the child’s symptoms began to disappear.

Megan Davenhall of Thinking Moms Revolution, mother of an 11-year-old boy with autism, told the EPA, “It’s going to be a long road for us.”

She began her research when her son was diagnosed at age three. As she turned to organic foods, and eliminated chemicals, he started to grow—something he hadn’t done for two and a half years. He weighed only 38 pounds at age six. Now, Davenhall told the EPA, “He’s doing better. He’s not off the spectrum. … It’s a long road for us, because my son was so very damaged. … He was skin and bones and it’s taken us years to recover his gut health.”

“The damage didn’t need to happen to him,” Davenhall said. “And I don’t want to see it happen to one other kid out there, not one. What we feed our kids, what we put into our bodies, is the most important thing. Healthy food should be available for everybody. It needs to happen. It needs to happen today.”

Sarah Cusack of Thriving Family Health talked about her daughter Claire who at 12 months, changed from a happy, easy-going baby to a miserable, constipated baby who was literally starving. She was emaciated. She had a huge bloated belly. At 20 months, she was diagnosed with celiac disease. But the turning point came when she switched to an organic diet. Claire is now a healthy six-year-old. Her mom is a health coach. Cusack says that an all-organic diet is the centerpiece of her practice. She’s seen improvement in clients with myriad health problems, including migraines, eczema, rashes, gastro-intestinal conditions, mood disorders like anxiety and depression, constipation and auto-immune conditions.

Swaying decision-makers with Science

After the testimonials, It was time to hit the EPA with hard science.

Honeycutt delivered a 20-minute presentation on how glyphosate figures as an environmental cause of so many of the diseases impacting our kids today. She left behind a binder, prepared by Moms Across America volunteers, packed with scientific articles supporting her assertions. Zen’s presentation and the materials she presented to the EPA covered the following points.

•    Exposure to glyphosate correlates with chronic illness. Chronically ill people have significantly higher levels of glyphosate in their systems than healthy people.

•    Glyphosate is an endocrine disruptor which is toxic to placental cells. This means it may impact our ability to conceive and carry healthy babies to term. It may also cause breast cancer.

•    Glyphosate destroys the gut bacteria we need for good health. Scientists have observed that in chickens and cattle, glyphosate kills the good gut bacteria while leaving behind bacteria that causes food poisoning. Glyphosate’s negative impact on our microbiome may be the reason for increasing rates of allergies, celiac sprue and gluten intolerance, and colitis and Crohn’s disease.

•    Glyphosate makes vaccines far more toxic than they would otherwise be. When children are overexposed to glyphosate, they are more likely to react badly to vaccination. There’s an intricate connection between the gut and the brain, such that an unhealthy digestive system translates into pathologies in the brain. Aluminum, mercury and glyphosate work synergistically to create severe deficiency in sulfate supplies to the brain. This may be what’s causing the epidemic levels of autism and other diseases such as Alzheimer’s.

•    Glyphosate is a chelator that deprives living things of vital nutrients, vitamins and minerals. This is how glyphosate kills plants. It may also be how it’s killing people. Glyphosate-induced vitamin deficiency may be a factor in the growing cancer rates. Glyphosate has been directly linked to Non-Hodgkin’s Lymphoma. A recent meta-analysis found that exposure to glyphosate doubled the likelihood of contracting B cell lymphoma.

Will the EPA consider this evidence and move to protect our children from glyphosate?

We’re about to find out.

For five years, the EPA has been collecting and analyzing data. This year (2014), the agency will publish a risk assessment and open a 60-day public comment period. Then it will publish a proposed registration and provide another opportunity for public comments.

Finally, the EPA will make a registration decision to either continue business-as-usual, place new restrictions on the use of glyphosate, or to take it off the market.

Moms want it off the market.

Moms Across America and Thinking Moms Revolution are currently working with the EPA to develop protocols for an independent scientific study of glyphosate in breast milk for inclusion in the agency’s review.

These moms won’t stop until Roundup is recalled and they need your help. Please join the Recall Roundup campaign at Moms Across America or Thinking Moms Revolution.

 

Alexis Baden-Mayer is political director of the Organic Consumers Association.For more information on this topic or related issues you can search the thousands of archived articles on the OCA website.Organic Consumers Association · 6771 South Silver Hill Drive, Finland MN 55603 ·
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Activist or Media Inquiries: 218-226-4164 · Fax: 218-353-7652 ·
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This is the article as it appears on the Organic Consumers Association site: http://www.organicconsumers.org/articles/article_30153.cfm

 

 

SOURCES CITED IN THE ARTICLE – listed in the order the links appear

All About Organics pageWhy We Should All Eat More Organic Food. Organic Consumers Association. See:  http://organicconsumers.org/organlink.cfm

Genetic Engineering page:  Earth Open Source. GMO Myths and Truths. Organic Consumers Association. See:  http://www.organicconsumers.org/gelink.cfm

Millions Against Monsanto pageMillions Against Monsanto. Organic Consumers Association. See: http://www.organicconsumers.org/monsanto/

Moms Across America:  Moms Across America. See: http://www.organicconsumers.org/monsanto/

Thinking Moms Revolution:  Thinking Moms Revolution. See: https://www.facebook.com/thinkingmomsrevolution

recall Monsanto’s Roundup:  See: https://www.facebook.com/events/897793350237253/

glyphosate:   Mercola, R. (April 15 2014). New Studies Reveal Damaging Effects of Glyphosate. Organic Consumers Association. See: http://www.organicconsumers.org/articles/article_29790.cfm

tested for glyphosate:  Glyphosate Testing Kit for Urine and Water. See: http://www.microbeinotech.com/Default.aspx?tabid=57

unsafe levels of glyphosate:  Glyphosate Testing Full Report: Findings in American Mothers’ Breast Milk, Urine and Water. See: http://www.momsacrossamerica.com/glyphosate_testing_results

Zen HoneycuttCA State Grange Rally for GE Labeling Jan 6th CA Capitol Steps- Zen Honeycutt’s Speech. Moms Across America. See: http://www.momsacrossamerica.com/ca_capitol_steps_speech

glyphosate and autismJeffrey Smith interviews Dr. Stephanie Seneff about Glyphosate. See video:  http://vimeo.com/6591412

GMO Free Lancaster County:  See:  https://www.facebook.com/GMOFREELANCASTERCOUNTYorg

linked to glyphosate exposure:   Samsel, A. & Seneff, S. (2013). Glyphosate’s Suppression of Cytochrome P450 Enzymes and Amino Acid Biosynthesis by the Gut Microbiome: Pathways to Modern Diseases. Entropy 2013, 15, 1416-1463. See: http://groups.csail.mit.edu/sls/publications/2013/Seneff_Entropy-15-01416.pdf

Thinking Moms Revolution:   Thinking Moms Revolution. See: http://thinkingmomsrevolution.com/

Thriving Family Health:  Sarah Cusack Scholl. See: http://www.thrivingfamilyhealth.com

Exposure to glyphosate:  Kruger, M. et al.(2014). Detection of Glyphosate Residues in Animals and Humans. Journal of Environmental & Analytical Toxicology, 2014, 4:2.
See: http://omicsonline.org/open-access/detection-of-glyphosate-residues-in-animals-and-humans-2161-0525.1000210.pdf

Glyphosate is an endocrine disrupter:   Gastnier, C. et al. (2009). Glyphosate-based herbicides are toxic and endocrine disruptors in human cell lines. Toxicology, 2009, 262:3, 184-91. See: http://www.ncbi.nlm.nih.gov/pubmed/19539684

toxic to placental cells:  Richard, S. et al. (2005). Differential Effects of Glyphosate and Roundup on Human Placental Cells and Aromatase. Environmental Health Perspectives, 2005; 113:6, 716–720. See: http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1257596/

ability to conceive:   Mercola, R. (April 1 2014). Roundup Toxicity May Impact Male Fertility: Study. See: http://articles.mercola.com/sites/articles/archive/2014/04/01/Roundup-toxicity-male-infertility.aspx

cause breast cancer:   Thongprakaisang S. et al. (2013). Glyphosate induces human breast cancer cells growth via estrogen receptors. Food & Chemical Toxicology, 2013, 59, 129-36. See: http://www.ncbi.nlm.nih.gov/pubmed/23756170

Glyphosate destroys the gut bacteria:  Sustainable Pulse. (Sept 7 2013). New Review Shows Glyphosate Destroys Human Health and Biodiversity. See: http://sustainablepulse.com/2013/09/07/new-review-shows-glyphosate-destroys-human-health-and-biodiversity/#.U4i24ighy-9

chickens:  Shehata. A. A. et al. (2013). The effect of glyphosate on potential pathogens and beneficial members of poultry microbiota in vitro. Current  Microbiology, 2013 66:4, 350-8. See: http://www.ncbi.nlm.nih.gov/pubmed/23224412

cattle:  Kruger, M. et al. (2013). Glyphosate suppresses the antagonistic effect of Enterococcus spp. on Clostridium botulinum. Anaerobe, 2013, 20,74-8.  See: http://www.ncbi.nlm.nih.gov/pubmed/23396248

Glyphsate’s negative impact on our microbiome:  Polan, M. (2013). Some of My Best Friends are Germs. New York Times Magazine, May 15 2013. See: http://www.nytimes.com/2013/05/19/magazine/say-hello-to-the-100-trillion-bacteria-that-make-up-your-microbiome.html?pagewanted=all

increasing rates of allergies:  Basulto, D. (2014). The secret to treating your allergies may lie in your stomach. WashingtonPost.com, April 17 2014. See: http://www.washingtonpost.com/blogs/innovations/wp/2014/04/17/the-secret-to-treating-your-allergies-may-lie-in-your-stomach/

celiac sprue and gluten intolerance:  Samsel, A. & Seneff, S. (2013). Glyphosate, pathways to modern diseases II: Celiac sprue and gluten intolerance. Interdisciplinary Toxicology, 2013, 6:4, 159–184. See: http://sustainablepulse.com/wp-content/uploads/2014/02/Glyphosate_II_Samsel-Seneff.pdf

Crohn’s disease:  Samsel, A. & Seneff, S. (2013). Glyphosate’s Suppression of Cytochrome P450 Enzymes and Amino Acid Biosynthesis by the Gut Microbiome: Pathways to Modern Diseases. Entropy, 2013, 15:4), 1416-1463. See: http://www.mdpi.com/1099-4300/15/4/1416

Glyphosate makes vaccines far more toxic:   Viadro, C. I. (2014). Sulfate, Sleep and Sunlight: The Disruptive and Destructive Effects of Heavy Metals and Glyphosate. Mercola.com. See:  http://articles.mercola.com/sites/articles/archive/2014/05/08/heavy-metals-glyphosate-health-effects.aspx

epidemic levels of autism and other diseases such as Alzheimer’s:  Seneff, S. (2013). Roundup: The “Nontoxic” Chemical that May Be Destroying our Health. The Weston A. Price Foundation. See:  http://www.westonaprice.org/health-topics/Roundup-the-nontoxic-chemical-that-may-be-destroying-our-health/

growing cancer rates:  Hardt, R. (2014). Glyphosate it binds minerals and cuts off the production of neurotransmitters and hormones: A visual connection of the routes of diseases and cancer. Academia.edu. See:  http://www.academia.edu/5772865/GLYPHOSATE_IT_BINDS_MINERALS_AND_CUTS_OFF_THE_PRODUCTION_OF_NEUROTRANSMITTERS_AND_HORMONES….A_VISUAL_CONNECTION_OF_THE_ROUTES_OF_DISEASES_AND_CANCER

exposure to glyphosate doubled the likelihood of contracting B cell lymphoma:  Schinasi, L. & Leon, M.E. (2014). Non-Hodgkin lymphoma and occupational exposure to agricultural pesticide chemical groups and active ingredients: a systematic review and meta-analysis. International Journal of Environmental Research and Public Health, 2014, 11:4, 4449-527. See:  http://www.ncbi.nlm.nih.gov/pubmed/24762670

Moms Across America:  Moms Across America. See:  https://www.facebook.com/MomsAcrossAmerica

Thinking Moms Revolution:  Thinking Moms Revolution. See:  https://www.facebook.com/thinkingmomsrevolution

Organic Consumers Association:  Organic Consumers Association. See: http://www.organicconsumers.org/

 

 

© Copyright 2014 Joan Rothchild Hardin. All Rights Reserved.

 

DISCLAIMER:  Nothing on this site or blog is intended to provide medical advice, diagnosis or treatment.